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Related Experiment Videos

The valve bladder: etiology and outcome.

Enrique Jaureguizar1, Pedro López-Pereira, M José Martinez-Urrutia

  • 1Department of Pediatric Urology, University Hospital La Paz, Paseo de la Castellana 261, 28046 Madrid, Spain. ejaureguizar@hulp.insalud.es.

Current Urology Reports
|June 27, 2002
PubMed
Summary

Posterior urethral valves cause irreversible bladder changes in boys, leading to common issues like bladder instability and myogenic failure. Treatment aims to improve kidney function and manage incontinence in affected patients.

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Area of Science:

  • Pediatric Urology
  • Developmental Biology
  • Nephrology

Background:

  • Posterior urethral valves (PUV) cause infravesical obstruction during fetal development.
  • This obstruction leads to structural, biochemical, and functional bladder alterations in affected boys.
  • These changes can be irreversible, impacting long-term bladder function.

Purpose of the Study:

  • To describe the bladder changes resulting from posterior urethral valves.
  • To identify common urodynamic patterns associated with PUV.
  • To outline treatment goals for bladder dysfunction in PUV patients.

Main Methods:

  • Review of structural, biochemical, and functional changes in bladders due to PUV.
  • Analysis of common urodynamic patterns observed in patients with PUV.

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  • Evaluation of treatment strategies for bladder dysfunction in PUV.

    • Main Results:

    • Common urodynamic patterns include bladder instability, poor compliance, and myogenic failure.
    • These patterns are linked to a poor long-term prognosis for some patients.
    • Bladder dysfunction significantly impacts kidney function and can lead to secondary urinary incontinence.

    Conclusions:

    • Infravesical obstruction from PUV induces significant, potentially irreversible, bladder changes.
    • Urodynamic findings like instability and myogenic failure are critical indicators of prognosis.
    • Management focuses on renal protection and addressing urinary incontinence.