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Expression of TFF1, TFF2 and TFF3 in gastric cancer

Hiroyuki Kirikoshi1, Masaru Katoh

  • 1Genetics and Cell Biology Section, Genetics Division, National Cancer Center Research Institute, Tsukiji 5-chome, Chuo-ku, Tokyo 104-0045, Japan.

Insights

Trefoil factor (TFF) gene expression, including TFF1, TFF2, and TFF3, was analyzed in human gastric cancer. TFF1 and TFF2 were often down-regulated, while TFF3 showed varied expression in gastric tumors.

Area of Science:

  • Oncology
  • Molecular Biology
  • Genetics

Background:

  • Estrogen regulates expression of trefoil factor (TFF) and WNT gene families.
  • WNT signaling molecules are crucial in various biological processes.
  • Gastric cancer development involves complex genetic alterations.

Purpose of the Study:

  • To investigate the expression profile of TFF1, TFF2, and TFF3 in human gastric cancer.
  • To analyze the differential expression of TFFs in gastric cancer cell lines and primary tumors.
  • To establish a comprehensive understanding of TFF expression in gastric cancer.

Main Methods:

  • Bioinformatics analysis, cDNA-library screening, and cDNA-PCR were used to characterize WNT signaling molecules.
  • Expression analysis of TFF1, TFF2, and TFF3 in seven human gastric cancer cell lines.
  • Quantitative analysis of TFF expression in twelve primary gastric cancer tissues.

Main Results:

  • TFF1, TFF2, and TFF3 showed differential expression patterns in gastric cancer cell lines.
  • TFFs were preferentially expressed in diffuse-type gastric cancer cell lines.
  • TFF1 and TFF2 were frequently down-regulated in primary gastric cancer (58.3% and 83.3%, respectively).
  • TFF3 exhibited down-regulation in 16.7% and up-regulation in 41.7% of primary gastric cancer cases.

Conclusions:

  • This study provides the first comprehensive expression analysis of TFFs in human gastric cancer.
  • TFF1 and TFF2 are frequently down-regulated in gastric cancer, suggesting a tumor-suppressive role.
  • TFF3 displays complex expression patterns, with potential roles in both suppression and promotion of gastric cancer.

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