[Chronic idiopathic gastric volvulus in children]

L Canazza1, E Bianchini, M Cerri

  • 1Istituti Clinici di Perfezionamento, Ospedale dei Bambini V. Buzzi, UO di Chirurgia Pediatrica, Via Castelvetro 32, 20154 Milano, Italia. chirpedbuzzi@tiscali.it

Insights

Chronic idiopathic volvulus, a gastric condition, may not always require surgery. Conservative treatments are effective for some patients, while others may need surgical intervention if symptoms persist or recur.

Area of Science:

  • Gastroenterology
  • Surgical Gastroenterology

Context:

  • Chronic idiopathic volvulus is a rare condition involving the abnormal rotation of the stomach.
  • Diagnosis can be challenging, especially with atypical symptoms mimicking gastroesophageal reflux disease.

Purpose:

  • To present clinical, radiographic, and instrumental data on 18 cases of gastric chronic idiopathic volvulus.
  • To evaluate the efficacy of conservative versus surgical management for this condition.

Summary:

  • 18 patients with gastric chronic idiopathic volvulus were diagnosed via barium meal or pHmetric esophageal monitoring.
  • Conservative treatment (antireflux positioning and prokinetics) was initiated, with 5 patients recovering after one year.
  • 13 patients required surgery due to lack of improvement or relapse during conservative therapy.

Impact:

  • Abnormal gastric position alone is not an absolute indication for surgery.
  • Surgical intervention should be considered when conservative medical management fails to alleviate symptoms or when relapse occurs.
Abstract

Related Concept Videos

Pyloric Obstruction01:11

Pyloric Obstruction

Pyloric obstruction, also referred to as gastric outlet obstruction, is a condition characterized by narrowing or blockage at the pylorus—the muscular valve regulating the flow of stomach contents into the duodenum. When this passage becomes impaired, the stomach cannot effectively empty its contents into the small intestine. This disruption leads to a range of gastrointestinal symptoms, including early satiety, bloating, epigastric pain, postprandial nausea, persistent vomiting, and...
Esophageal Achalasia01:27

Esophageal Achalasia

Esophageal achalasia is a chronic neurogenic disorder characterized by impaired relaxation of the lower esophageal sphincter (LES) and absent or ineffective peristalsis in the distal esophagus. This leads to a functional obstruction without a physical blockage, despite significant disruption of esophageal motility.EtiologyAchalasia is caused by degeneration of the myenteric (Auerbach's) plexus, specifically the loss of inhibitory ganglion cells that produce vasoactive intestinal peptide (VIP)...
Peptic Ulcer01:27

Peptic Ulcer

Peptic ulcers are erosive lesions of the gastric or duodenal lining, most commonly caused by Helicobacter pylori infection. This Gram-negative, helical bacterium has adapted to survive the stomach’s acidic environment by producing urease, which converts urea into ammonia and carbon dioxide. The ammonia neutralizes gastric acid in the bacterium’s immediate environment, allowing colonization of the gastric mucosa. H. pylori attaches to mucus-secreting epithelial cells, penetrates the mucus...
Gastritis II: Pathophysiology01:26

Gastritis II: Pathophysiology

The pathophysiology of gastritis begins with the colonization of the stomach lining by Helicobacter pylori (H. pylori). This bacterium spreads mainly via the oral-oral route through saliva or shared utensils, and can also be transmitted in overcrowded or unhygienic environments through contaminated water, despite its brief survival outside the body.ColonizationOnce ingested, H. pylori enters the stomach and begins colonization by navigating through the mucus layer lining the stomach wall. It...
Peptic Ulcer Disease V: Surgical Management and Nursing Care01:25

Peptic Ulcer Disease V: Surgical Management and Nursing Care

Surgical management and nursing care are crucial in treating Peptic Ulcer Disease (PUD). Here is an organized and enhanced overview of the surgical interventions and the associated nursing care for PUD:
Surgical Interventions for Peptic Ulcer Disease
Esophageal Varices-I: Introduction01:24

Esophageal Varices-I: Introduction

Esophageal varices are dilated, tortuous veins which are found mainly in the submucosa of the lower esophagus but which may also appear higher up or extend into the stomach. They develop due to increased pressure in the portal venous system, often as a result of liver cirrhosis. This condition scars and damages the liver, impeding normal blood flow through the portal vein. To compensate, blood seeks alternative pathways, forming fragile new vessels (varices) in the esophagus and stomach. These...