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[Hypophosphatemias (author's transl)].

J P Knochel

    La Nouvelle Presse Medicale
    |January 13, 1979
    PubMed
    Summary
    This summary is machine-generated.

    Severe hypophosphatemia, a condition of low blood phosphate, can occur due to antacid use or in critical illness. Prompt treatment with phosphate supplements is crucial to prevent serious complications like hemolysis and neurological issues.

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    Area of Science:

    • Biochemistry
    • Internal Medicine
    • Critical Care Medicine

    Context:

    • Hypophosphatemia can arise from specific antacid interactions with intestinal phosphorus.
    • It is also observed in severe burn patients during refeeding after malnutrition.
    • Other contexts include intense hyperventilation, diabetic ketoacidosis, and alcoholism.

    Purpose:

    • To outline the causes and clinical manifestations of hypophosphatemia.
    • To describe the potential hematologic, platelet, and neurologic complications.
    • To emphasize the importance of timely and adequate phosphate repletion.

    Summary:

    • Ingestion of certain antacids can lead to severe hypophosphatemia by binding phosphorus in the gut.
    • This electrolyte imbalance is associated with erythrocyte abnormalities, impaired platelet function, and neurological disturbances.

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  • Treatment involves prompt administration of sodium or potassium phosphate to maintain blood levels above 10 mg/L.

    • Impact:

    • Highlights the significant risks of hypophosphatemia in various clinical settings.
    • Underscores the necessity of early diagnosis and intervention to prevent severe patient outcomes.
    • Provides clear treatment guidelines for managing this critical condition.