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Secretion of basic fibroblast growth factor (FGF-2) by WEHI-3B myelomonocytic leukemia cells

Augusto Pessina1, Giuseppina Gagliardi, Cristina Croera

  • 1Institute of Microbiology, University of Milan, Via Pascal 36, 20133 Milan, Italy. pessinaa@mailserver.unimi.it

Insights

Fibroblast Growth Factors (FGFs) are expressed in leukemia and stromal cells. Secreted FGF-2 from leukemia cells stimulates stromal cell growth, suggesting FGFs support leukemogenesis.

Area of Science:

  • Hematology
  • Molecular Biology
  • Cancer Research

Background:

  • Fibroblast Growth Factors (FGFs) are crucial signaling molecules.
  • Their role in hematopoietic and stromal cells, particularly in leukemia, requires further investigation.

Purpose of the Study:

  • To investigate the expression and function of various FGFs (FGF-1 to FGF-6) in murine leukemia (WEHI-3B) and stromal (SR-4987) cell lines.
  • To determine the role of secreted FGF-2 in supporting stromal cell function and its potential contribution to leukemogenesis.

Main Methods:

  • Reverse Transcription Polymerase Chain Reaction (RT-PCR) for mRNA expression analysis.
  • Affinity chromatography, chromatofocusing, and immunoblotting for FGF-2 detection in cell culture supernatants.
  • Agar clonogenic assay to assess the biological activity of FGF-2 on stromal cells.

Main Results:

  • Both WEHI-3B and SR-4987 cells express mRNA for FGF-1, FGF-2, and FGF-6.
  • WEHI-3B cells also express mRNA for FGF-3 and FGF-4.
  • Supernatant from WEHI-3B cells significantly enhanced the agar clonogenicity of SR-4987 cells, mediated by secreted FGF-2.
  • SR-4987 cells express FGF-2 mRNA but do not appear to secrete it.

Conclusions:

  • FGFs, including oncogene-related factors like FGF-3, FGF-4, and FGF-6, are expressed in myeloid leukemia cells.
  • Secreted FGF-2 from leukemia cells positively regulates bone marrow stromal cell function.
  • FGFs likely play a significant role in sustaining the leukemogenic process and related hematological disorders.

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