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Neuropathological, biochemical and genetic alterations in AD

P H St George-Hyslop1, J McLaurin, P E Fraser

  • 1Centre for Research in Neurodegenerative Diseases, Crescent West, University of Toronto, Toronto, Ontario, Canada.

Drug News & Perspectives
|August 26, 2003
PubMed
Summary

Abnormal processing of beta-amyloid precursor protein (beta APP) is key in Alzheimer's disease (AD) pathogenesis. Current research focuses on modulating amyloid beta (A beta) production or inhibiting its aggregation to treat AD.

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