Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Ocular motor dysfunction in stupor and coma.

U W Buettner1

  • 1Department of Neurology, Eberhard-Karls University, Tübingen, Germany.

Bailliere'S Clinical Neurology
|August 1, 1992
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

[Chronic pain therapy].

Praxis·2009
Same author

Orofacial cluster headache.

Cephalalgia : an international journal of headache·2008
Same author

[Neurophysiological aspects of chronic pain].

Praxis·2005
Same author

Multiple visual hallucinations and pseudohallucinations in one individual patient: when the world is turning upside down and the television keeps falling to the ground while dwarfs are parading on the ceiling.

Ophthalmologica. Journal international d'ophtalmologie. International journal of ophthalmology. Zeitschrift fur Augenheilkunde·2005
Same author

Meningoradiculitis associated with giant cell arteritis.

Neurology·2002
Same author

The contribution of the vertical semicircular canals to high-velocity horizontal vestibulo-ocular reflex (VOR) in normal subjects and patients with unilateral vestibular nerve section.

Acta oto-laryngologica·1996
Same journal

Remyelination in demyelinating disease.

Bailliere's clinical neurology·1999
Same journal

Other immunomodulatory therapies in multiple sclerosis.

Bailliere's clinical neurology·1999
Same journal

Copolymer-1.

Bailliere's clinical neurology·1999
Same journal

Interferon beta 1a.

Bailliere's clinical neurology·1999
Same journal

Treatment of multiple sclerosis with interferon beta 1b.

Bailliere's clinical neurology·1999
Same journal

The cause and management of bladder, sexual and bowel symptoms in multiple sclerosis.

Bailliere's clinical neurology·1999
See all related articles

Ocular motor disorders in stupor and coma provide key clinical signs. Assessing eye movements helps determine brainstem lesion sites and document deficits for better patient care.

Area of Science:

  • Neurology
  • Ophthalmology

Background:

  • Ocular motor disorders are significant clinical indicators in stupor and coma.
  • Understanding these signs aids in diagnosing neurological conditions.

Purpose of the Study:

  • To elucidate the pathophysiological mechanisms of ocular motor dysfunction in stupor and coma.
  • To emphasize the importance of precise documentation of ocular motor deficits.

Main Methods:

  • Clinical observation and bedside maneuvers.
  • Elicitation of reflex eye movements (e.g., vestibulo-ocular reflex via head rotation or caloric irrigation).

Main Results:

  • Many ocular motor signs, including conjugate eye deviations and spontaneous movements, have clear pathophysiological bases.

Related Experiment Videos

  • Vestibulo-ocular reflex (VOR) assessment can differentiate between intact VOR and defective gaze-holding mechanisms.
  • Conclusions:

    • Accurate documentation of ocular motor deficits, including pupil status, eye position, spontaneous movements, and VOR, is crucial.
    • Bedside assessment of ocular motor function aids in localizing brainstem lesions and understanding neurological impairment.