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Updated: Jul 11, 2026

Preterm EEG: A Multimodal Neurophysiological Protocol
Published on: February 18, 2012
Transient neonatal hypothyroxinemia and the auditory brainstem evoked response
D Kohelet1, E Arbel, M Goldberg
1Department of Neonatology, Assaf Harofeh Medical Center, Zerifin, Israel.
Abstract:
Ten premature infants with low serum thyroxine levels (less than 84 nmol/L) were compared with 10 biochemically euthyroid infants to determine whether hypothyroxinemia in such infants can lead to alterations in the auditory pathway. The auditory pathway was studied between 6 and 11 wk of life by analyzing brainstem evoked potentials elicited by a 10/s, 75 dB above normal adult hearing level click stimulus presented at the infant's ears. Peak latencies of components I, III, and V and interpeak latencies I-III, III-V, and I-V did not yield statistically significant differences between groups. The present study indicates that untreated neonatal hypothyroxinemia does not lead to abnormalities of auditory brainstem evoked response.
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