Related Experiment Videos
[Rademaker and Garcin syndrome associated with pallidal calcifications]
Revue Neurologique
|January 1, 1992
Summary
This study details gait abnormalities in an elderly woman, characterized by exaggerated antigravity support. Findings suggest reflexive abnormalities and basal ganglia involvement in postural control.
Area of Science:
- Neurology
- Gait Analysis
- Neurophysiology
Background:
- Gait abnormalities can stem from various neurological conditions.
- Exaggerated antigravity support is a rare gait disturbance.
- Previous research linked this to cerebellar and brainstem lesions.
Observation:
- A 76-year-old woman presented with gait abnormalities and exaggerated antigravity support.
- Quantitative locomotion analysis revealed reduced stride duration and length.
- Increased variability, stance duration, and double-support duration were observed.
Findings:
- Electromyography demonstrated leg hypertension upon ankle dorsiflexion.
- This suggests reflexive antigravity support abnormalities, distinct from other gait disorders.
- Bilateral pallidal calcifications were noted, implicating basal ganglia.
Implications:
- The basal ganglia may play a role in antigravity support and postural muscle tone.
- Understanding these mechanisms is crucial for diagnosing and managing gait disorders.
- This case expands the known pathophysiology of gait disturbances.