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Interactions between kinematics and loading during walking for the normal and ACL deficient knee
Thomas P Andriacchi1, Chris O Dyrby
1Department of Mechanical Engineering, Division of Biomechanical Engineering, Stanford University, Stanford, CA 94305-4038, USA. tandriac@stanford.edu
Journal of Biomechanics
|December 16, 2004
Summary
Anterior cruciate ligament (ACL) deficiency alters knee motion during walking, particularly in the swing phase. Muscle adaptations may compensate, but altered knee alignment can lead to degenerative joint changes.
Area of Science:
- Biomechanics
- Orthopedics
- Sports Medicine
Background:
- Anterior cruciate ligament (ACL) injuries are common, leading to knee instability and potential long-term joint degeneration.
- Understanding knee kinematics after ACL deficiency is crucial for developing effective rehabilitation and treatment strategies.
Purpose of the Study:
- To identify specific walking cycle phases where extrinsic knee forces cause abnormal kinematics in ACL-deficient knees.
- To correlate altered knee kinematics with joint loading and potential degenerative changes.
Main Methods:
- Compared intersegmental forces and moments with tibial motion (translation and rotation) relative to the femur during key walking phases.
- Analyzed kinematic changes in ACL-deficient knees during heel strike, weight acceptance, terminal extension, and swing phases.
Main Results:
- ACL deficiency primarily caused abnormal knee kinematics during the terminal swing phase, characterized by reduced tibial external rotation and anterior translation.
- A consistent internal rotation offset of the tibia was observed throughout the gait cycle in ACL-deficient knees.
- This rotational offset correlated with the flexion moment during weight acceptance, suggesting quadriceps muscle compensation.
Conclusions:
- Muscle activation patterns can adapt to compensate for altered knee kinematics following ACL loss.
- Altered tibiofemoral contact due to abnormal rotation may contribute to meniscus and articular cartilage degeneration after ACL injury.