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Are small for gestational age infants at higher risk for intracranial lesions?

M E Sims1, C Troy, F J Walther

  • 1Department of Pediatrics, Los Angeles County-University of Southern California Medical Center, University of Southern California School of Medicine.

Insights

Small for gestational age (SGA) infants do not show increased central nervous system (CNS) abnormalities compared to appropriate for gestational age (AGA) neonates. Cranial ultrasounds revealed no significant differences in CNS abnormalities between the two groups.

Area of Science:

  • Neonatal Neurology
  • Pediatric Radiology
  • Fetal Development

Background:

  • The relationship between small for gestational age (SGA) and central nervous system (CNS) abnormalities is debated.
  • Existing research presents conflicting findings, with some suggesting higher risks and others proposing protective effects of growth retardation.

Purpose of the Study:

  • To investigate if intrauterine growth retardation in SGA neonates is associated with a higher incidence of CNS abnormalities detectable by cranial ultrasound.
  • To compare cranial ultrasound findings between SGA and appropriate for gestational age (AGA) neonates.

Main Methods:

  • A comparative study involving 197 SGA infants and 197 matched AGA neonates.
  • Cranial ultrasounds were performed to detect CNS abnormalities.
  • Infants with external dysmorphology or TORCH infections were excluded from the analysis.

Main Results:

  • No significant difference in the overall incidence of intracranial abnormalities was found between SGA (26%) and AGA (21%) infants.
  • Hemorrhagic or ischemic lesions, the most common abnormalities, occurred with similar frequency in both groups (SGA: 22%, AGA: 18%).
  • The type and severity of lesions, as well as non-hemorrhagic ventriculomegaly and structural lesions, were comparable between SGA and AGA infants.

Conclusions:

  • SGA infants, when free from external dysmorphology and TORCH infections, do not exhibit an elevated risk for CNS abnormalities detectable by cranial ultrasound.
  • The findings suggest that growth retardation itself may not predispose neonates to CNS damage detectable by this imaging modality.

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