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Bartter's syndrome--case report.

A Miyoshi, H Makino, M Hiramatsu

    Acta Medica Okayama
    |August 1, 1979
    PubMed
    Summary
    This summary is machine-generated.

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    This case report details a patient with Bartter

    Area of Science:

    • Endocrinology and Nephrology
    • Thyroid Disorders
    • Electrolyte Imbalances

    Background:

    • Graves' disease is an autoimmune disorder affecting the thyroid gland.
    • Bartter's syndrome is a rare kidney disorder causing electrolyte imbalances.
    • Co-occurrence of these conditions presents diagnostic and management challenges.

    Observation:

    • A 26-year-old female with a history of Graves' disease presented with persistent fatigue and polyuria.
    • Despite normalized thyroid function with methimazole treatment, hypokalemia persisted.
    • Clinical evaluation revealed hypokalemic alkalosis, elevated plasma renin activity, and high aldosterone levels.

    Findings:

    • The patient exhibited normal pressor response to norepinephrine but diminished sensitivity to angiotensin II.

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  • Renal biopsy demonstrated juxtaglomerular cell hyperplasia.
  • Electron microscopy confirmed proliferation of lacis cells (agranular cells) in the juxtaglomerular apparatus.

    • Implications:

    • This case highlights a potential association between Graves' disease and Bartter's syndrome.
    • The findings suggest a possible link between thyroid autoimmunity and secondary hyperaldosteronism.
    • Further research is warranted to elucidate the pathophysiology of this rare co-occurrence.