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Further evidence of increased polymorphonuclear cell activity in patients with Raynaud's phenomenon

C S Lau1, A B Bridges, A Muir

  • 1University Department of Medicine, Ninewells Hospital and Medical School, Dundee.

Insights

Polymorphonuclear cells (PMNs) are more active in patients with Raynaud's phenomenon (RP), indicating their role in vascular disease. This heightened PMN activity may contribute to the condition

Area of Science:

  • Vascular pathophysiology
  • Immunology
  • Rheumatology

Background:

  • Polymorphonuclear cells (PMNs) play a role in ischaemic vascular disease.
  • Previous studies suggest increased white cell activity in Raynaud's syndrome (RS).
  • The link between underlying conditions, digital vasospasm, and PMN activity in RS is unclear.

Purpose of the Study:

  • To assess polymorphonuclear cell (PMN) activity in patients with severe Raynaud's phenomenon (RP).
  • To investigate the relationship between PMN activity and digital vasospasm severity in RP.
  • To measure plasma malondialdehyde (MDA) levels as an indicator of free radical activity.

Main Methods:

  • A whole blood PMN aggregation assay was used to measure PMN activity.
  • 38 patients with severe RP (16 with systemic sclerosis and secondary RS, 22 with RP alone) were studied.
  • Plasma MDA levels were measured, and results were compared to 56 matched volunteers. Patients recorded Raynaud's attack frequency and duration.

Main Results:

  • Patients with RP, with or without systemic sclerosis (SSc), exhibited significantly greater reductions in single PMN count compared to controls.
  • SSc-associated RS showed a 48.2% fall (P=0.04), and RP alone showed a 49.3% fall (P=0.01) in single PMN count versus controls.
  • Correlation analysis assessed the relationship between clinical variables of Raynaud's severity and white cell activity.

Conclusions:

  • Elevated PMN activity is a significant finding in patients with Raynaud's phenomenon.
  • This heightened PMN activity may be directly related to the pathophysiology of RP, independent of underlying conditions like SSc.
  • Further research is warranted to elucidate the precise mechanisms by which PMNs contribute to RP and ischaemic vascular disease.

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