Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

New developments in occupational asthma.

P S Burge1

  • 1Occupational Lung Disease Unit, East Birmingham Hospital, UK.

British Medical Bulletin
|January 1, 1992
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Occupational asthma in office workers.

Occupational medicine (Oxford, England)·2022
Same author

Occupational asthma caused by peracetic acid-hydrogen peroxide mixture.

Occupational medicine (Oxford, England)·2019
Same author

Cleaning agent occupational asthma in the West Midlands, UK: 2000-16.

Occupational medicine (Oxford, England)·2018
Same author

Asbestosis is prevalent in a variety of construction industry trades.

NPJ primary care respiratory medicine·2018
Same author

Occupational asthma caused by acrylic compounds from SHIELD surveillance (1989-2014).

Occupational medicine (Oxford, England)·2017
Same author

Biopsy-proven hypersensitivity pneumonitis caused by a fluorocarbon waterproofing spray.

Occupational medicine (Oxford, England)·2017
Same journal

Human health in relation to visible, functional, and accessible green space: a systematic review and narrative synthesis of the 3 + 30 + 300 guideline.

British medical bulletin·2026
Same journal

Muscle herniae in exercise-induced leg pain: diagnostic pitfalls and the 'repair paradox'.

British medical bulletin·2026
Same journal

Scaffolds and platelet concentrates in bone regenerative medicine: applications, mechanisms, and future approaches.

British medical bulletin·2026
Same journal

Back squat and deadlift fatiguing protocols elicit distinct countermovement jump profiles: phase-specific predictors and soreness responses.

British medical bulletin·2026
Same journal

Enhancing bone healing through osteogenic medium components: biological roles, mechanisms, and clinical applications.

British medical bulletin·2026
Same journal

Moral injury in healthcare workers: causes & interventions.

British medical bulletin·2026
See all related articles

Occupational asthma affects high-risk groups, with incidence tied to exposure agents and levels. Smoking and bronchial responsiveness are significant factors, and prevention is key.

Area of Science:

  • Occupational Medicine
  • Pulmonology
  • Immunology

Background:

  • Occupational asthma is a significant workplace health concern.
  • Recent data provide independent estimates of its incidence in the general working population.
  • High-risk occupations, such as those involving isocyanates and flour/wheat exposure, are consistently identified.

Purpose of the Study:

  • To review recent developments in occupational asthma.
  • To examine factors influencing incidence, including exposure, sensitisation, and smoking.
  • To discuss the relationship between bronchial responsiveness and occupational asthma.

Main Methods:

  • Review of independent incidence estimates.
  • Analysis of factors influencing occupational asthma development.

Related Experiment Videos

  • Examination of the role of specific IgE and bronchial responsiveness.
  • Discussion of outcomes following exposure cessation.

    • Main Results:

    • Incidence of occupational asthma is primarily determined by the specific agent and level of exposure.
    • Small molecular weight chemicals that bind to body proteins appear crucial.
    • Smoking increases the risk of developing both specific IgE and occupational asthma.
    • Pre-existing bronchial hyperresponsiveness may predispose individuals to occupational asthma, or occupational sensitization may cause it.
    • Approximately 20% of affected workers have normal baseline measurements.
    • Delayed removal from exposure often leads to persistent asthma.

    Conclusions:

    • Occupational asthma incidence is linked to specific exposures and individual factors like smoking and bronchial responsiveness.
    • Understanding these factors is crucial for prevention strategies.
    • Effective prevention and timely intervention are essential to manage and mitigate occupational asthma.