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Characteristics of conjugative R-plasmids from pathogenic avian Escherichia coli

R E Wooley1, K R Spears, J Brown

  • 1Department of Medical Microbiology, College of Veterinary Medicine, University of Georgia, Athens 30602.

Avian Diseases
|April 1, 1992
PubMed

Insights

Virulent avian Escherichia coli transferred R-plasmids conferring antibiotic resistance to recipient strains. However, virulence factors like colicin production and embryo lethality were not transferred, suggesting R-plasmids are not linked to avian E. coli virulence.

Area of Science:

  • Microbiology
  • Bacterial genetics
  • Avian pathogens

Background:

  • Avian pathogenic Escherichia coli (E. coli) pose a significant threat to poultry health.
  • Understanding the genetic basis of E. coli virulence and antibiotic resistance is crucial for effective disease management.
  • R-plasmids are known to carry antibiotic resistance genes and can be transferred between bacterial strains.

Purpose of the Study:

  • To investigate the role of R-plasmids in the virulence of avian E. coli isolates.
  • To determine if specific virulence factors are co-transferred with antibiotic resistance genes on R-plasmids.
  • To assess the association between R-plasmids and key virulence traits in avian E. coli.

Main Methods:

  • Isolation and characterization of virulent avian E. coli strains.
  • Plasmid transfer experiments using conjugation to recipient E. coli strains.
  • Assessment of antibiotic resistance profiles before and after plasmid transfer.
  • Evaluation of virulence factor transfer, including colicin production, siderophore production, complement resistance, and embryo lethality.

Main Results:

  • Three of four virulent avian E. coli isolates successfully transferred a large R-plasmid to recipient strains.
  • Transferred R-plasmids conferred resistance to streptomycin and a combination of streptomycin-tetracycline-sulfa.
  • Crucially, virulence-associated traits such as colicin production, siderophore production, complement resistance, and embryo lethality were not transferred with the R-plasmids.

Conclusions:

  • The R-plasmids studied in these virulent avian E. coli isolates are primarily associated with antibiotic resistance.
  • The genetic determinants for the observed virulence factors in these strains appear to be located elsewhere on the chromosome or on different mobile genetic elements.
  • These findings suggest that R-plasmid-mediated antibiotic resistance is not directly linked to the virulence of these specific avian E. coli isolates.

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