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Zenker's diverticulum and reflux.

H Feussner1, J R Siewert

  • 1Chirurgische Klinik und Poliklinik TUM, Klinikum rechts der Isar, München, Germany.

Hepato-Gastroenterology
|April 1, 1992
PubMed
Summary
This summary is machine-generated.

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Gastroesophageal reflux disease (GER) may contribute to Zenker's diverticulum, but evidence suggests muscle changes in the upper esophageal sphincter are more likely causes. Further research is needed to clarify the exact relationship between GER and Zenker's diverticulum.

Area of Science:

  • Gastroenterology
  • Otolaryngology
  • Esophageal Motility Disorders

Background:

  • The relationship between gastroesophageal reflux disease (GERD) and Zenker's diverticulum (ZD) is not fully understood.
  • Epidemiological data suggest a link, as ZD is more prevalent in populations with high GERD incidence.

Purpose of the Study:

  • To investigate the potential causal link between GERD and the development of ZD.
  • To explore alternative etiological factors for ZD, focusing on upper esophageal sphincter (UES) function.

Main Methods:

  • Review of existing physiological and pathophysiological investigations.
  • Analysis of clinical observations and surgical outcomes in patients with ZD.
  • Examination of the role of GERD as a risk factor in ZD development and post-myotomy complications.

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Main Results:

  • Current evidence does not definitively prove GERD as a cause of ZD.
  • Emerging data suggest that intrinsic changes in UES muscle motility may be a more significant factor.
  • Clinical experience indicates GERD plays a minor role in individual ZD cases, with low complication rates even without preoperative reflux screening.

Conclusions:

  • While GERD might contribute to ZD in some instances, autochthonous structural lesions in the UES muscle fibers are more probable causes in most cases.
  • Other unidentified factors may also contribute to the pathogenesis of Zenker's diverticulum.