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Molecular cloning and expression of the type 1 and type 2 murine receptors for tumor necrosis factor

R G Goodwin1, D Anderson, R Jerzy

  • 1Immunex Corporation, Seattle, Washington 98101.

Insights

Researchers isolated murine tumor necrosis factor (TNF) receptors, finding they mirror human receptor binding characteristics and map to different chromosomes. This advances understanding of TNF signaling pathways.

Area of Science:

  • Immunology
  • Molecular Biology
  • Genetics

Background:

  • Tumor necrosis factor (TNF) receptors mediate crucial cellular responses.
  • Understanding the structure and function of TNF receptors is vital for immunology and disease research.

Purpose of the Study:

  • To isolate and characterize murine tumor necrosis factor (TNF) receptors.
  • To compare murine TNF receptors with their human counterparts.
  • To determine the chromosomal locations of murine TNF receptor genes.

Main Methods:

  • Cross-hybridization using probes from cloned human TNF receptors to isolate murine receptor clones.
  • Sequence homology analysis between murine and human receptors.
  • Ligand-binding assays with recombinant murine receptors.
  • RNA analysis to study transcript expression.
  • Chromosomal mapping of TNF receptor genes using murine cDNAs.

Main Results:

  • Murine type 1 (p80) and type 2 (p60) TNF receptors were isolated, showing ~65% amino acid identity to human homologs.
  • Recombinant murine receptors exhibited similar ligand-binding characteristics to human receptors, binding TNF-alpha and -beta with cross-competition.
  • Transcripts for both receptor types were found in all examined murine cells.
  • Murine TNF receptor genes were mapped to chromosomes 4 (type 1) and 6 (type 2), indicating they are not linked.

Conclusions:

  • Murine TNF receptors share significant sequence homology and functional characteristics with human receptors.
  • The differential chromosomal localization of murine TNF receptor genes provides insights into their genetic regulation.
  • These findings contribute to a deeper understanding of TNF signaling pathways and their genetic basis.

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