Related Experiment Video

Updated: Aug 7, 2026

Permanent Cerebral Vessel Occlusion via Double Ligature and Transection
08:22

Permanent Cerebral Vessel Occlusion via Double Ligature and Transection

Published on: July 21, 2013

Complications of migraine: migrainous infarction

Giorgio Bono1, Giorgio Minonzio, Marco Mauri

  • 1Universitary Center for Adaptive Disorders and Headache, Dipartimento di Medicina Clinica, Università degli Studi dell'Insubria, Varese, Italy. neurova@tin.it

Clinical and Experimental Hypertension (New York, N.Y. : 1993)
|July 13, 2006
PubMed

Insights

Migrainous infarction (MI) is a rare complication of migraine. Extended diagnostic workup is crucial to differentiate MI from other cerebrovascular disorders and identify risk factors in migraine patients.

Area of Science:

  • Neurology
  • Vascular Neurology
  • Neuroscience

Background:

  • Migraine and cerebrovascular disorders frequently coexist.
  • Clinical symptom overlap presents diagnostic and therapeutic challenges.
  • Migrainous infarction (MI) exemplifies the complex relationship between migraine and stroke.

Purpose of the Study:

  • To discuss diagnostic criteria for migrainous infarction.
  • To explore epidemiological and clinicopathogenetic implications of MI.
  • To emphasize the importance of ruling out other conditions in suspected MI cases.

Main Methods:

  • Review of diagnostic criteria for migrainous infarction.
  • Analysis of epidemiological studies on MI.
  • Discussion of clinicopathogenetic factors.

Main Results:

  • Migrainous infarction is a rare complication of migraine.
  • Extended diagnostic workup is necessary for "possible" MI cases.
  • Conditions like patent foramen ovale and thrombophilia are critical risk factors for stroke in migraineurs.

Conclusions:

  • Accurate diagnosis of migrainous infarction requires excluding symptomatic migraine due to vascular pathology.
  • Identifying and considering precipitating factors is essential.
  • Further research into the interplay between migraine and cerebrovascular events is warranted.

More Related Videos

Brain Infarct Segmentation and Registration on MRI or CT for Lesion-symptom Mapping10:25

Brain Infarct Segmentation and Registration on MRI or CT for Lesion-symptom Mapping

Published on: September 25, 2019

Transient Middle Cerebral Artery Occlusion Model of Stroke05:32

Transient Middle Cerebral Artery Occlusion Model of Stroke

Published on: August 11, 2023

Related Concept Videos

Ischemic Stroke ll: Pathophysiology01:15

Ischemic Stroke ll: Pathophysiology

An ischemic stroke occurs when a cerebral blood vessel becomes obstructed, most often by a thrombus or embolus, interrupting the delivery of oxygen and glucose to brain tissue. Because neurons rely on continuous aerobic metabolism, energy failure begins within minutes of reduced perfusion. The region receiving the least blood flow becomes the infarct core, an area of irreversible cellular death. Surrounding this core lies the penumbra, a zone of hypoperfused but still viable tissue that is...
Ischemic Stroke l: Introduction01:15

Ischemic Stroke l: Introduction

Ischemic stroke is an acute cerebrovascular condition in which blood flow to a brain region is suddenly interrupted, leading to tissue infarction. Neurons depend on continuous oxygen and glucose supply, so even brief reductions in perfusion cause energy failure, ionic imbalance, and irreversible injury. Ischemic strokes are classified into thrombotic and embolic types based on their underlying mechanisms.Thrombotic MechanismsThrombotic stroke develops when a clot forms within a cerebral artery.
Transient Ischemic Attack l: Introduction01:26

Transient Ischemic Attack l: Introduction

A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...
Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Hemorrhagic Stroke l: Introduction01:17

Hemorrhagic Stroke l: Introduction

A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
Increased Intracranial Pressure ll: Pathophysiology01:29

Increased Intracranial Pressure ll: Pathophysiology

Increased intracranial pressure (ICP) refers to a potentially life-threatening rise in pressure inside the skull. This usually happens when there is a major change in the volume of brain tissue, blood, or cerebrospinal fluid (CSF) — the three components inside the skull. According to the Monro-Kellie doctrine, if the volume of one component increases, the volumes of the other components must decrease to maintain normal pressure. If this does not happen, ICP rises.The process often begins with...