Related Experiment Videos
Early MRI and outcomes of untreated patients with mild or improving ischemic stroke
V Rajajee1, C Kidwell, S Starkman
1UCLA Medical Center, Los Angeles, CA, USA. vrajajee@yahoo.com
Objective:
To determine the frequency of early neurologic deterioration with infarct expansion (ENDIE) and poor outcomes among ischemic stroke patients not treated with reperfusion therapies because of rapidly improving or mild symptoms (RIMS) and to study the predictive value of hyperacute MRI in these patients.
Methods:
We identified consecutive patients with symptoms of acute stroke undergoing multimodal MRI within 6 hours of onset without evidence of hemorrhage on imaging. Medical records were reviewed for evidence of early neurologic deterioration within 48 hours. All deteriorating patients had repeat MRI to ascertain causes of worsening. Poor outcome was defined as a discharge modified Rankin Scale (mRS) score of > or = 3.
Results:
We identified 74 patients with stroke symptoms < or = 6 hours from onset. Forty had RIMS, and 39 did not receive reperfusion therapies because of RIMS. Among these 39, 4 experienced ENDIE, and 8 were discharged with mRS score of > or = 3. Eight of the 39 patients had large-vessel occlusions on MR angiography. Three of 8 patients with large-vessel occlusion as against only one of 31 patients without occlusion had ENDIE (odds ratio [OR] 18, 95% CI 1.6 to 209, p = 0.02). Four of 8 patients with large-vessel occlusion as against 4 of 31 patients without occlusion had a discharge mRS score of > or = 3 (OR 7, 95% CI 1.2 to 38, p = 0.04).
Conclusions:
About 10% of patients eligible for acute reperfusion therapy excluded on the basis of mild or rapidly improving symptoms show early neurologic deterioration with infarct expansion within 48 hours, and about 20% show poor outcome at discharge. Persisting large-vessel occlusion substantially increases the risk of early worsening and poor functional outcome.
Insights
Approximately 10% of ischemic stroke patients with mild or rapidly improving symptoms experience early neurologic deterioration, and 20% have poor outcomes. Large-vessel occlusions significantly increase these risks, highlighting the need for careful assessment even in seemingly mild stroke cases.
Area of Science:
- Neurology
- Radiology
- Stroke Medicine
Background:
- Ischemic stroke patients with rapidly improving or mild symptoms (RIMS) are often excluded from reperfusion therapies.
- The risk of early neurologic deterioration with infarct expansion (ENDIE) and poor outcomes in this specific patient group is not well-defined.
- Hyperacute magnetic resonance imaging (MRI) may offer predictive value in assessing these patients.
Purpose of the Study:
- To determine the frequency of ENDIE and poor outcomes in ischemic stroke patients not receiving reperfusion due to RIMS.
- To investigate the predictive value of hyperacute MRI in identifying patients at risk for ENDIE and poor outcomes.
Main Methods:
- Consecutive acute ischemic stroke patients underwent multimodal MRI within 6 hours of symptom onset.
- Patients were assessed for ENDIE within 48 hours, with repeat MRI for deteriorating cases.
- Poor outcome was defined as a modified Rankin Scale (mRS) score of ≥3 at discharge.
Main Results:
- Among 39 patients not treated with reperfusion due to RIMS, 4 experienced ENDIE, and 8 had poor outcomes (mRS ≥3).
- Large-vessel occlusions (LVO) were present in 8 patients, significantly increasing the risk of ENDIE (OR 18) and poor outcome (OR 7) compared to those without LVO.
- Hyperacute MRI identified LVO, a key predictor of adverse events.
Conclusions:
- About 10% of ischemic stroke patients eligible for reperfusion but excluded due to RIMS experience ENDIE within 48 hours.
- Approximately 20% of these patients have a poor functional outcome at discharge.
- Persisting large-vessel occlusion is a substantial risk factor for early worsening and poor functional outcome in RIMS patients.
Related Concept Videos
Imaging Studies for Cardiovascular System IV: CMRI
Ischemic Stroke l: Introduction
Ischemic Stroke ll: Pathophysiology