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Subcutaneous Trigeminal Nerve Field Stimulation for Refractory Facial Pain
Published on: May 10, 2017
[Trigeminal autonomic cephalgias: diagnostic and therapeutic implications]
Mirjam Rosenberg-Nordmann1, Thomas R Tölle, Till Sprenger
1Neurologische Klinik und Poliklinik der Technischen Universität München, Klinikum rechts der Isar, München. nmirjam@gmx.net
MMW Fortschritte Der Medizin
|October 20, 2007
Summary
Trigeminal autonomic cephalgias (TACs) are severe headache syndromes with facial autonomic symptoms. While sharing similar mechanisms, cluster headache (CH), paroxysmal hemicrania (PH), and SUNCT require different treatments, and structural lesions can impact management.
Area of Science:
- Neurology
- Headache Medicine
- Neuroscience
Background:
- Trigeminal autonomic cephalgias (TACs) encompass severe primary headache disorders.
- TACs include cluster headache (CH), paroxysmal hemicrania (PH), and short-lasting neuralgiform headache with conjunctival injection and tearing (SUNCT).
- Cluster headache is the most prevalent TAC syndrome.
Purpose of the Study:
- To summarize the characteristics of TACs.
- To highlight the differing pathophysiologic mechanisms and therapeutic strategies among TACs.
- To underscore the clinical significance of structural lesions in TAC management.
Main Methods:
- Literature review of TACs.
- Analysis of pathophysiologic mechanisms.
- Comparison of therapeutic approaches.
- Review of cases involving structural lesions.
Main Results:
- TACs share hypothalamic and trigeminovascular involvement.
- Therapeutic strategies vary significantly across TAC subtypes.
- Structural lesions, though uncommon, necessitate altered treatment plans.
Conclusions:
- Understanding TAC heterogeneity is crucial for effective treatment.
- Distinct management protocols are required for CH, PH, and SUNCT.
- Identification of structural lesions is vital for optimizing patient care in TACs.
