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Mucosubstance histochemistry of the maxillary sinus mucosa in experimental sinusitis: a model study on rabbits

K M Westrin1, P Stierna, B Carlsöö

  • 1Department of Otorhinolaryngology, Huddinge Hospital, Sweden.

Insights

Experimental sinusitis in rabbits altered sinus mucosubstances, increasing secretory cells and goblet cells containing specific mucins. These changes were observed regardless of Streptococcus pneumoniae inoculation.

Area of Science:

  • Histochemistry
  • Pathology
  • Microbiology

Background:

  • Sinusitis involves inflammation of the sinus mucosa.
  • Mucosubstances play a crucial role in mucosal defense.
  • Understanding mucosubstance changes in sinusitis is important for disease management.

Purpose of the Study:

  • To investigate the histochemical properties of mucosubstances in experimental sinusitis in rabbits.
  • To compare changes in rabbits with ostial blockage versus those with additional bacterial inoculation.

Main Methods:

  • Induction of experimental sinusitis in rabbits via maxillary sinus ostium occlusion.
  • Optional inoculation with Streptococcus pneumoniae.
  • Histochemical analysis of sinus mucosa using periodic acid-Schiff and Alcian blue stains.

Main Results:

  • Increased periodic acid-Schiff positive epithelial secretory cells observed.
  • Goblet cell regeneration occurred from the second week.
  • Goblet cells contained periodate-reactive acid mucins and periodate-unreactive acid mucins, with varying proportions of sialomucins and sulphomucins.
  • Submucosal and nasal glands showed no significant histochemical changes.
  • No distinct differences in staining patterns between occlusion and pneumococcal groups.

Conclusions:

  • Experimental sinusitis induces significant changes in sinus mucosal mucosubstances, particularly in epithelial secretory cells and goblet cells.
  • The observed mucin changes are primarily related to the inflammatory process rather than the specific bacterial challenge.
  • Histochemical alterations in mucosubstances may contribute to the pathophysiology of sinusitis.

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