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[Alternative localization of the determinant of pathogenicity coded by the Yersinia pseudotuberculosis pVM82 plasmid]

Insights

Epidemiologically hazardous Yersinia pseudotuberculosis strains possess chromosomal DNA homologous to immunosuppression-encoding plasmid pVM82. This genetic difference correlates with suppressed antibody formation in these virulent bacterial strains.

Area of Science:

  • Microbiology
  • Immunology
  • Genetics

Background:

  • Yersinia pseudotuberculosis is an opportunistic pathogen.
  • Plasmid pVM82 confers immunosuppressive properties to bacteria.
  • The genetic basis for immunosuppression in virulent strains is not fully understood.

Purpose of the Study:

  • To investigate the chromosomal DNA regions in Yersinia pseudotuberculosis homologous to the immunosuppression-encoding plasmid pVM82.
  • To compare the distribution of these homologous regions in virulent and nonvirulent strains.
  • To confirm the immunosuppressive activity of virulent strains.

Main Methods:

  • DNA hybridization using probes from the 25 Md DNA segment of plasmid pVM82.
  • Analysis of chromosomal DNA dispersion patterns in different Yersinia pseudotuberculosis strains.
  • Assessment of antibody formation suppression in response to Yersinia pseudotuberculosis antigens.

Main Results:

  • Chromosomal DNA regions homologous to the 25 Md segment of pVM82 were identified in Yersinia pseudotuberculosis.
  • The dispersion of these homologous regions differed between epidemiologically hazardous and nonvirulent strains.
  • Epidemiologically hazardous strains demonstrated suppression of antibody formation to key antigens, similar to strains with pVM82.

Conclusions:

  • Virulent Yersinia pseudotuberculosis strains harbor chromosomal DNA with homology to the immunosuppression-mediating plasmid pVM82.
  • This genetic homology is associated with the observed immunosuppressive phenotype in hazardous strains.
  • The findings suggest a chromosomal mechanism for immunosuppression in Yersinia pseudotuberculosis.

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