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Molecular action of tricholin, a ribosome-inactivating protein isolated from Trichoderma viride

A Lin1, C K Chen, Y J Chen

  • 1Institute of Genetics, National Yang-Ming Medical College, Taipei, Taiwan, Republic of China.

Molecular Microbiology
|December 1, 1991
PubMed

Insights

A novel fungal protein, tricholin, inhibits protein synthesis by targeting ribosomes. This protein, isolated from Trichoderma viride, mimics alpha-sarcin activity by cleaving ribosomal RNA.

Area of Science:

  • Biochemistry
  • Molecular Biology
  • Mycology

Background:

  • Soil-borne fungi like Trichoderma viride produce various bioactive compounds.
  • Protein synthesis is a fundamental cellular process regulated by numerous factors.
  • Ribosomes are the cellular machinery responsible for protein synthesis.

Purpose of the Study:

  • To isolate and characterize an extracellular protein from Trichoderma viride.
  • To investigate the inhibitory effects of the isolated protein on cell-free protein synthesis.
  • To elucidate the mechanism of action of the protein, particularly its interaction with ribosomes.

Main Methods:

  • Isolation and purification of an extracellular protein from Trichoderma viride.
  • Determination of the protein's amino acid composition and molecular mass.
  • Assay of the protein's effect on rabbit reticulocyte lysate protein synthesis.
  • Analysis of ribosomal RNA cleavage products and cross-reactivity with alpha-sarcin antibodies.

Main Results:

  • An acidic extracellular protein, named tricholin, with a molecular mass of 14,200 daltons was isolated.
  • Tricholin potently inhibited cell-free protein synthesis in rabbit reticulocyte lysate at 6.3 x 10(-7) M.
  • Tricholin induced cleavage of ribosomal RNA, generating an alpha-sarcin RNA fragment, indicating ribosome interaction.
  • Antibodies against alpha-sarcin showed strong cross-reactivity with tricholin, and vice versa.

Conclusions:

  • Tricholin is a novel, potent inhibitor of protein synthesis originating from Trichoderma viride.
  • The mechanism of inhibition involves direct interaction with ribosomes, leading to ribosomal RNA cleavage.
  • Tricholin's activity and immunological cross-reactivity suggest functional and structural similarities to alpha-sarcin.

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