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Are TNF-alpha and IL-1 beta relevant in the pathogenesis of migraine without aura?

V Covelli1, I Munno, N M Pellegrino

  • 1Clinica Neurologica, II Facoltà di Medicina e Chirurgia, Università di Napoli, Italy.

Acta Neurologica
|April 1, 1991
PubMed

Insights

Migraine without aura is linked to immune system dysfunction. Studies show elevated tumor necrosis factor-alpha (TNF-alpha) in patients, potentially due to bacterial lipopolysaccharides (LPS).

Area of Science:

  • Immunology
  • Neurology
  • Pathophysiology

Background:

  • Migraine without aura (MWA) is associated with immune deficits.
  • Patients with MWA show impaired phagocytosis and killing by polymorphonuclear cells (PMN) and monocytes.
  • This immune dysfunction may explain increased infection rates in MWA patients.

Purpose of the Study:

  • To investigate the presence of circulating cytokines (CKs) in individuals with MWA.
  • To explore the correlation between immune dysfunction and MWA pathogenesis.

Main Methods:

  • Evaluation of spontaneous cytokine release in MWA subjects.
  • Detection of bacterial lipopolysaccharides (LPS) in plasma samples.

Main Results:

  • An exaggerated spontaneous release of tumor necrosis factor-alpha (TNF-alpha) was observed in MWA individuals.
  • Detectable levels of bacterial lipopolysaccharides (LPS) were found in the plasma of these patients.
  • A correlation was established between elevated TNF-alpha and LPS presence.

Conclusions:

  • Circulating TNF-alpha, potentially triggered by LPS, may play a role in MWA pathogenesis.
  • The neuro-vascular and demyelinating effects of TNF-alpha warrant further investigation in MWA.
  • Immune dysregulation is a significant factor in the development of migraine without aura.

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