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The effect of aspirin on Gomori-positive glia in mouse brain
Piotr Sura1, Zbigniew Srebro, Bogdan Wiliński
1Departament of Human Developmental Biology, Collegium Medicum, Jagiellonian University, Kopernika 7, 31-034 Kraków, Poland. mbsura@cyf-kr.edu.pl
Abstract:
Aspirin (acetylsalicylic acid, ASA) treatment resulted in a significant decrease in the amount of the sulfur-rich Gomori-positive material present in the cytoplasm of periventricular glia. It also caused the accumulation of the Gomori-positive neurosecretory material in the supraoptic and paraventricular nuclei and, most pronounced, in the neurosecretory axons of the paraventricular- and supraoptic-neurophypophysial tract.
