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Related Experiment Video

Updated: Jun 24, 2026

Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model

Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model

Published on: June 18, 2021

Traumatic brainstem hemorrhage presenting with hemiparesis.

Young Bem Se¹, Choong Hyun Kim, Koang Hum Bak

¹Department of Neurosurgery, Hanyang University Guri Hospital, Guri, Korea.

Journal of Korean Neurosurgical Society

|April 9, 2009

Summary

Traumatic brainstem hemorrhage is rare after head injury. Magnetic resonance imaging is crucial for diagnosing brainstem bleeds when other injuries don't explain neurological deficits.

Keywords:

Brain stem hemorrhage Head trauma Hemiparesis

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Massive Pontine Hemorrhage by Dual Injection of Autologous Blood

Massive Pontine Hemorrhage by Dual Injection of Autologous Blood

Published on: May 29, 2021

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Last Updated: Jun 24, 2026

Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model

Pre-Chiasmatic, Single Injection of Autologous Blood to Induce Experimental Subarachnoid Hemorrhage in a Rat Model

Published on: June 18, 2021

Massive Pontine Hemorrhage by Dual Injection of Autologous Blood

Massive Pontine Hemorrhage by Dual Injection of Autologous Blood

Published on: May 29, 2021

Area of Science:

Neurotraumatology
Neuroradiology

Background:

Traumatic brainstem hemorrhage (TBH) is an infrequent yet serious consequence of blunt head trauma.
The rostral midline brainstem is the typical location for TBH, associated with a poor prognosis due to the critical anatomical location.

Observation:

A case study involving a 41-year-old male with drowsiness and right hemiparesis post-blunt head injury.
Initial imaging (skull radiographs, CT scans) revealed superficial injuries (skull fracture, epidural hematoma, contusion) insufficient to explain the hemiparesis.
Cervical spine MRI identified a pontomedullary junction lesion, confirmed by brain MRI (diffusion-weighted and FLAIR sequences) showing ventral pontomedullary hyperintensity, more pronounced on the left.

Findings:

The pontomedullary junction lesion on MRI correlated with the patient's neurological deficits, particularly hemiparesis.
Conservative management led to progressive improvement in the patient's mental status and motor weakness.

Implications:

Highlights the importance of considering brainstem hemorrhage in head-injured patients with unexplained neurological deficits.
Suggests advanced neuroimaging, such as MRI, is essential for diagnosing less common traumatic brain injuries like TBH.
Emphasizes that TBH, even when rare, should be part of the differential diagnosis for neurological impairments following head trauma.