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Transient Ischemic Attack l: Introduction

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A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...

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A Murine Model of Ischemic Retinal Injury Induced by Transient Bilateral Common Carotid Artery Occlusion

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Transient cortical blindness after coronary angiography.

B N Alp¹, N Bozbuğa, M A Tuncer

¹Department of Health, Education Faculty, Marmara University, Istanbul, Turkey.

The Journal of International Medical Research

|September 19, 2009

Summary

Transient cortical blindness is a rare complication following coronary angiography. This case highlights a patient who experienced temporary vision loss after the procedure, with spontaneous recovery.

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Area of Science:

Neurology
Radiology
Cardiology

Background:

Coronary angiography is a common diagnostic procedure.
Transient cortical blindness (TCB) is a rare adverse event associated with contrast media.
Previous contrast exposure does not preclude TCB.

Observation:

A 56-year-old patient developed TCB 72 hours post-coronary angiography.
Neurological exam revealed cortical blindness without focal deficits.
Brain imaging showed bilateral occipital lobe contrast enhancement on CT, with normal MRI.

Findings:

The patient experienced temporary visual impairment following contrast administration.
Contrast enhancement in the occipital lobes suggests a contrast-related mechanism.
Spontaneous visual recovery occurred within four days.

Implications:

This case underscores the potential for delayed TCB after coronary angiography.
Radiologists and cardiologists should be aware of this rare complication.
Further research may elucidate the pathophysiology and risk factors for contrast-induced TCB.