Related Experiment Video
Updated: Jun 17, 2026

07:45
"Sun's Seven-Step Technique" for Endoscopic En-Bloc Resection of Thyroid Cancer via the Chest-Breast Approach
Published on: November 28, 2025
Thyroid gland hematoma after blunt neck trauma
Baris Saylam1, Bülent Comçali, Mehmet Vasfi Ozer
1Ankara Numune Teaching and Research Hospital, Department of 3rd Surgery, Ankara, Turkey.
The Western Journal of Emergency Medicine
|January 5, 2010
Summary
Blunt trauma can rarely cause acute thyroid gland hemorrhage, leading to hoarseness. This case highlights diagnosis via laryngoscopy, CT, and angiography, with successful conservative management.
Area of Science:
- Endocrinology
- Trauma Surgery
- Diagnostic Imaging
Background:
- Thyroid gland hemorrhage from blunt trauma is exceptionally rare.
- Acute thyroid hematomas can present with airway compromise symptoms like hoarseness.
Purpose of the Study:
- To report a rare case of blunt trauma-induced thyroid hemorrhage.
- To describe the diagnostic approach and successful conservative management of this condition.
Main Methods:
- Case report of a patient with blunt neck trauma.
- Diagnostic workup included fiberoptic laryngoscopy, cervical computed tomography (CT), and carotid angiography.
- Treatment involved conservative management.
Main Results:
- The patient presented with hoarseness secondary to acute thyroid hemorrhage.
- Diagnostic imaging confirmed a thyroid gland hematoma.
- Conservative treatment resulted in a favorable outcome without complications.
Conclusions:
- Blunt trauma can precipitate thyroid gland hemorrhage, a rare clinical entity.
- A combination of laryngoscopy and advanced imaging is crucial for diagnosis.
- Conservative management can be effective for thyroid hematomas in select cases.
Related Concept Videos
Goiter
Goiter refers to an abnormal enlargement of the thyroid gland that may appear as a diffuse goiter (uniform enlargement) or nodular (single or multiple nodules). Functionally, it is classified as nontoxic (normal/low hormone levels) or toxic (excess hormone production).PathophysiologyDiffuse thyroid enlargement typically results from prolonged stimulation by thyroid-stimulating hormone (TSH) or TSH-like agents, commonly seen in hypothyroidism or iodine deficiency. In contrast, in hyperthyroid...
Graves Disease II: Pathophysiology
Graves’ disease is an autoimmune disorder characterized by the production of thyroid-stimulating immunoglobulins (TSI) that activate TSH receptors, leading to excessive synthesis and release of thyroid hormones (T3 and T4) and resulting in hyperthyroidism.Among all causes of hyperthyroidism, Graves’ disease is the most common and can happen at any age, though it is more frequent in women. It produces a hypermetabolic state with features such as weight loss, tachycardia, tremor, and heat...
The Thyroid Gland
The thyroid gland is a small, butterfly-shaped gland located in the neck and covers the anterior surface of the trachea. The gland has two lateral lobes connected by a thin tissue mass called the isthmus. Internally, each lobe comprises many small spherical structures known as thyroid follicles, surrounded by a network of blood vessels.
The follicles have a central cavity lined by simple cuboidal to squamous epithelial cells called follicular cells. These cells produce the glycoprotein...
The follicles have a central cavity lined by simple cuboidal to squamous epithelial cells called follicular cells. These cells produce the glycoprotein...
Hyperthyroidism II: Pathophysiology
Hyperthyroidism is a hypermetabolic state caused by elevated levels of thyroid hormones, triiodothyronine (T3) and thyroxine (T4). It results from dysregulation at the thyroid, pituitary, or immune system level and affects multiple organ systems.PathophysiologyThe most common cause of hyperthyroidism is Graves’ disease, an autoimmune disorder in which antibodies, specifically thyroid-stimulating antibodies (TSAb), a subtype of TSH receptor antibodies (TRAb), bind to and activate TSH receptors...
Hyperthyroidism I: Introduction
Hyperthyroidism is a type of thyrotoxicosis characterized by the thyroid gland's overproduction of the thyroid hormones triiodothyronine (T3) and thyroxine (T4). This hormone excess increases the basal metabolic rate and enhances sensitivity to catecholamines.DiagnosisDiagnosis is based on clinical features and biochemical testing. It typically shows suppressed thyroid-stimulating hormone (TSH) levels below 0.4 mIU/L, with elevated free T3 and/or T4. Additional tests, including thyroid...
Graves' Disease I: Introduction
Graves' disease is an autoimmune disorder that causes hyperthyroidism, or overactivity of the thyroid gland. It results from autoantibodies called thyroid-stimulating immunoglobulins (TSIs), which bind to thyroid-stimulating hormone (TSH) receptors, leading to overstimulation of hormone production and a hypermetabolic state.EtiologyAlthough considered idiopathic, Graves’ disease has well-established contributing factors. There is a strong genetic component, with increased prevalence in...