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A Modified Trier Social Stress Test for Vulnerable Mexican American Adolescents
Published on: July 10, 2017
Depressive symptoms, trait aggression, and cardiovascular reactivity to a laboratory stressor
Julia D Betensky1, Richard J Contrada
1Department of Psychology, Rutgers, The State University of New Jersey, 53 Avenue E, Piscataway, NJ 08854-8040, USA.
Insights
Depression and verbal aggression interact to increase blood pressure reactivity in women. This interaction may contribute to mechanisms underlying heart disease development.
Area of Science:
- Psychology
- Cardiology
- Behavioral Medicine
Background:
- Depression and anger/aggression traits are linked to coronary heart disease via stress-related cardiovascular processes.
- The interplay between these traits and cardiovascular reactivity (CVR) remains unclear, with possibilities including redundancy, addition, or synergy.
Purpose of the Study:
- To investigate the independent and interactive effects of depressive symptoms and anger/aggression traits on CVR.
- To determine how these psychological factors influence physiological responses to stress.
Main Methods:
- 63 healthy female participants completed self-report questionnaires for depression and aggression.
- Participants underwent a stressful speaking task while systolic blood pressure (SBP), diastolic blood pressure (DBP), and heart rate were monitored.
Main Results:
- A significant interaction was found between depressive symptoms and verbal aggression.
- Depressive symptoms were associated with increased SBP and DBP reactivity in women with high verbal aggression, but not in those with low verbal aggression.
Conclusions:
- Depressive symptoms and verbal aggression may synergistically promote exaggerated blood pressure responses to stressors.
- These exaggerated responses could be a marker for mechanisms contributing to heart disease.
Background:
Depression and anger/aggression-related traits are thought to promote coronary disease, at least in part, through their associations with stress-related cardiovascular processes. It is unclear whether the effects of these factors on cardiovascular reactivity (CVR) are redundant, additive, or synergistic.
Purpose:
The main goal of this study was to examine the independent and interactive effects of depressive symptoms and anger/aggression-related traits in promoting CVR.
Methods:
Participants were 63 healthy females who completed the Beck Depression Inventory and Buss-Perry Aggression Questionnaire and performed a stressful speaking task. Systolic and diastolic blood pressure (SBP and DBP) and heart rate were recorded.
Results:
Significant interaction effects indicated that depressive symptoms were positively associated with SBP and DBP reactivity among women high in verbal aggression but not among those low in verbal aggression.
Conclusions:
Depressive symptoms and verbal aggression may interact to promote exaggerated blood pressure responses to stressors, a possible marker for mechanisms that contribute to heart disease.
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