Related Experiment Video
Updated: Jun 12, 2026

Assessing Forelimb Function after Unilateral Cervical SCI using Novel Tasks: Limb Step-alternation, Postural Instability and Pasta Handling
Published on: September 16, 2013
Lower extremity monoparesis after aneurysmal subarachnoid hemorrhage
Dean Kostov1, Brian Jankowitz, Hilal Kanaan
1Department of Neurosurgery, University of Pittsburgh, Pittsburgh, PA 15213, USA. kostovdb@upmc.edu
Subarachnoid hemorrhage from ruptured aneurysms can cause delayed leg weakness. This painless neuropathy may stem from nerve root irritation due to blood in the lumbar cistern.
Area of Science:
- Neurology
- Neurosurgery
- Vascular Neurology
Background:
- Acute subarachnoid hemorrhage (SAH) is a critical neurological emergency.
- Ruptured intracranial aneurysms are a common cause of SAH.
- Endovascular coil embolization is a standard treatment for intracranial aneurysms.
Observation:
- Two patients treated for ruptured intracranial aneurysms developed delayed lower extremity monoparesis.
- The monoparesis was painless and resolved spontaneously over several months.
- No other neurological symptoms were associated with the monoparesis.
Findings:
- Extensive investigations were performed to determine the cause of the monoparesis.
- The leading hypothesis is peripheral neuropathy resulting from nerve root irritation.
- Irritation is attributed to the presence of abundant subarachnoid blood in the lumbar cistern.
Implications:
- This finding suggests a potential complication of subarachnoid hemorrhage treatment.
- Understanding this mechanism may improve patient monitoring and management strategies.
- Further research is warranted to confirm this hypothesis and its prevalence.
Related Concept Videos
Peripheral Artery Disease V: Postoperative Nursing Management
Peripheral Arterial Disease II: Clinical Manifestations and Diagnostic Evaluation
Peripheral Artery Disease III: Interprofessional Care
Secondary Spinal Cord Injury llI: Pathophysiology