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Related Experiment Video

Updated: Jun 10, 2026

An Effective Mouse Model of Unilateral Renal Ischemia-Reperfusion Injury

An Effective Mouse Model of Unilateral Renal Ischemia-Reperfusion Injury

Published on: July 15, 2021

[Mesenteric ischemia following a left radical nephrectomy].

M Demesmaeker¹, V Argo, A Tardieu

¹Service d'urologie, hôpital Tenon, 4, rue de la Chine, 75020 Paris, France. matdemes@hotmail.com

Progres En Urologie : Journal De L'Association Francaise D'Urologie Et De La Societe Francaise D'Urologie

|July 27, 2010

Summary

Nephrectomy in dialysis patients poses increased vascular risks. This case highlights multifactorial mesenteric ischemia post-nephrectomy, involving hypovolemia and crystal deposits.

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Last Updated: Jun 10, 2026

An Effective Mouse Model of Unilateral Renal Ischemia-Reperfusion Injury

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Controlled Reversible Visceral Arterial Ischemia, Venous Congestion and Combined Malperfusion via Midline Laparotomy in Rats

Controlled Reversible Visceral Arterial Ischemia, Venous Congestion and Combined Malperfusion via Midline Laparotomy in Rats

Published on: July 5, 2024

Area of Science:

Nephrology
Vascular Surgery
Gastroenterology

Background:

Nephrectomy is common in dialysis patients.
These patients face elevated vascular complication risks.
Mesenteric ischemia is a rare but serious post-nephrectomy complication.

Observation:

A patient developed mesenteric ischemia after left nephrectomy.
The ischemia was linked to splanchnic hypoperfusion and microvascular occlusion.

Findings:

Multifactorial causes included post-operative hypovolemia from dialysis fluid depletion.
Use of vasoactive amines contributed to splanchnic hypoperfusion.
Kayexalate crystal deposits caused mesenteric microvascular occlusion.

Implications:

Highlights the need for careful fluid management in dialysis patients undergoing nephrectomy.
Suggests awareness of kayexalate's potential role in mesenteric vascular events.
Emphasizes the complex interplay of factors leading to ischemia in this patient population.