Related Experiment Videos
Chronic expanding hematoma after spinal surgery
Hiroyuki Tsuchie1, Naohisa Miyakoshi, Michio Hongo
1Department of Orthopedic Surgery, Akita University Graduate School of Medicine, Akita 010-8543, Japan. tsuchie@doc.med.akita-u.ac.jp
The Spine Journal : Official Journal of the North American Spine Society
|September 21, 2010
Summary
A rare case of chronic expanding hematoma (CEH) occurred 24 years after spinal surgery. This delayed CEH highlights the importance of considering long-term complications following spine procedures.
Area of Science:
- Neurosurgery
- Spinal Surgery
- Hematology
Background:
- Chronic expanding hematoma (CEH) is an exceptionally rare complication following spinal surgery.
- No prior cases of CEH have been documented in the literature occurring decades after initial spinal intervention.
Observation:
- A 71-year-old male presented with a gradually enlarging spinal mass 24 years post-thoracic laminectomy and tumor resection.
- The mass, first noted 5 years after surgery, progressively grew over 19 years, causing erosion of thoracic vertebrae.
Findings:
- Surgical resection of the mass confirmed a chronic expanding hematoma.
- Histopathology verified the diagnosis, with no recurrence observed at the 1-year follow-up.
- Vertebral erosion was noted, but the dura mater remained intact.
Implications:
- This case underscores that chronic expanding hematoma can manifest as a very late complication of spinal surgery.
- Clinicians should consider CEH in the differential diagnosis for spinal masses, irrespective of the time elapsed since the original surgery.
Related Concept Videos
Increased Intracranial Pressure l: Introduction
Intracranial hypertension is a sustained elevation of intracranial pressure (ICP) above 22 mm Hg. In supine adults, normal ICP is ~7–15 mm Hg.The rigid, nonexpandable cranium contains three components—brain tissue, blood, and cerebrospinal fluid (CSF)—that total ~1,700 mL in a typical adult: 1,400 mL brain (~80%), 150 mL blood (~10%), and 150 mL CSF (~10%). According to the Monro–Kellie doctrine, total intracranial volume is effectively fixed. When one component expands, CSF and venous blood...
Hemorrhagic Stroke ll: Pathophysiology
A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Increased Intracranial Pressure ll: Pathophysiology
Increased intracranial pressure (ICP) refers to a potentially life-threatening rise in pressure inside the skull. This usually happens when there is a major change in the volume of brain tissue, blood, or cerebrospinal fluid (CSF) — the three components inside the skull. According to the Monro-Kellie doctrine, if the volume of one component increases, the volumes of the other components must decrease to maintain normal pressure. If this does not happen, ICP rises.The process often begins with...
Cerebral Edema ll: Pathophysiology
Vasogenic edema is a major form of cerebral edema characterized by abnormal accumulation of fluid in the brain’s extracellular space due to disruption of the blood–brain barrier (BBB). The BBB is a specialized structure composed of endothelial cells connected by tight junctions, supported by astrocytic endfeet and a basement membrane. Under normal conditions, it tightly regulates the movement of ions, proteins, and solutes between the bloodstream and brain parenchyma. When this barrier loses...
Hemorrhagic Stroke l: Introduction
A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
Brain Abscess l: Introduction
A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...