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Updated: Jun 8, 2026

Fixed Volume or Fixed Pressure: A Murine Model of Hemorrhagic Shock

Fixed Volume or Fixed Pressure: A Murine Model of Hemorrhagic Shock

Published on: June 6, 2011

A PRESsing dissection.

Tamika M Burrus¹, Bahram Mokri, Alejandro A Rabinstein

¹Mayo Clinic, Rochester, MN, USA. tamika.burrus@ucsf.edu

Neurocritical Care

|October 5, 2010

Summary

This summary is machine-generated.

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Carotid dissection can cause baroreceptor reflex failure, leading to severe hypertension. This case highlights how this can result in posterior reversible encephalopathy syndrome (PRES).

Area of Science:

Neurology
Vascular Neurology
Neurocritical Care

Background:

Intracranial dissections are linked to baroreceptor reflex dysfunction.
Baroreceptor failure can precipitate labile hypertension and associated neurological complications.
Posterior reversible encephalopathy syndrome (PRES) is a recognized complication of severe hypertension.

Observation:

This report details a patient who experienced carotid dissection.
The dissection led to a failure of the baroreceptor reflex.
The patient subsequently developed PRES.

Findings:

Carotid artery dissection was identified as the cause of baroreceptor reflex failure.
The resulting hypertension was directly linked to the dissection.

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Last Updated: Jun 8, 2026

Fixed Volume or Fixed Pressure: A Murine Model of Hemorrhagic Shock

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Published on: June 6, 2011

Isolation and Excision of Murine Aorta; A Versatile Technique in the Study of Cardiovascular Disease

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This hypertension precipitated the development of PRES.

Implications:

This case underscores the critical role of the baroreceptor reflex in maintaining hemodynamic stability after carotid dissection.
Early recognition of baroreceptor dysfunction is crucial for managing hypertension and preventing PRES.
Understanding this pathway may inform therapeutic strategies for similar neurological emergencies.