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Age-related changes in biotransformation of azoxymethane and methylazoxymethanol in vitro

T F McMahon1, J O Peggins, M M Centra

  • 1Department of Pharmacology and Toxicology, University of Maryland, Baltimore.

Insights

Aging reduces the liver

Area of Science:

  • Toxicology
  • Biochemistry
  • Gerontology

Background:

  • Azoxymethane (AZO) is a colon carcinogen.
  • Age-related metabolic changes can affect carcinogen biotransformation and risk.
  • Fischer 344 rats are a common model for aging studies.

Purpose of the Study:

  • To investigate age-related changes in the metabolism of azoxymethane (AZO) and its metabolite methylazoxymethanol (MAM) in male Fischer 344 rats.
  • To determine the role of hepatic hydroxylation and colonic Phase I metabolism in AZO/MAM biotransformation across different age groups.
  • To explore the potential involvement of colonic glucuronyltransferase in MAM biotransformation.

Main Methods:

  • Comparison of hepatic AZO to MAM conversion and colonic MAM metabolism by alcohol dehydrogenase (ADH) in young, middle-aged, and old male Fischer 344 rats.
  • Measurement of hepatic cytochrome P-450 content.
  • Assessment of MAM inhibition of colonic 4-methylumbelliferone glucuronyltransferase activity.

Main Results:

  • A significant decrease in hepatic conversion of AZO to MAM was observed in old rats compared to young rats.
  • Hepatic cytochrome P-450 content also decreased with age.
  • No age-related differences were found in the colonic metabolism of MAM by ADH.
  • MAM exhibited non-competitive inhibition of colonic glucuronyltransferase, suggesting it is not a substrate for this enzyme.

Conclusions:

  • Ageing alters AZO and MAM biotransformation in male Fischer 344 rats, specifically reducing hepatic hydroxylation.
  • Reduced hepatic metabolism of AZO to MAM in older rats may lead to decreased carcinogenic potential.
  • Colonic glucuronyltransferase is unlikely to be involved in the conjugation of MAM.

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