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Sources of propylene glycol and glycol ethers in air at home
Hyunok Choi1, Norbert Schmidbauer, John Spengler
1Department of Environmental Health, Harvard School of Public Health, USA. hchoi@hsph.harvard.edu
Insights
Common household activities like cleaning and repainting are significant sources of indoor propylene glycol and glycol ethers (PGEs). High humidity can prolong PGE presence, impacting children's respiratory health.
Area of Science:
- Environmental Health
- Indoor Air Quality
- Pediatric Health
Background:
- Propylene glycol and glycol ethers (PGEs) in indoor air are linked to childhood asthma, allergies, and sensitization.
- Identifying sources of PGEs is crucial for mitigating health risks in children's environments.
Purpose of the Study:
- To investigate the sources of PGEs in the indoor air of homes with preschool-aged children.
- To analyze the relationship between building characteristics, cleaning habits, and PGE concentrations.
Main Methods:
- A study of 390 Swedish homes examined building features, humidity, air exchange, and collected air/dust samples.
- Analysis included quantifying 16 PGEs, terpene hydrocarbons, Texanols, and phthalates (BBzP, DEHP).
- Building inspectors assessed water damage, mold odor, structural factors, temperature, and humidity.
Main Results:
- Home cleaning (mopping), recent repainting, and new bedroom surface materials were major contributors to PGE levels.
- Elevated indoor humidity prolonged PGE presence in the air post-painting.
- Water-based cleaning was linked to terpene levels, but no significant predictors were found for Texanols.
Conclusions:
- Household activities and building factors significantly influence indoor PGE concentrations.
- High humidity plays a role in sustained PGE levels, potentially impacting children's health.
- Disparate sources of indoor pollutants support previous findings on PGEs and respiratory conditions.
Abstract:
Propylene glycol and glycol ether (PGE) in indoor air have recently been associated with asthma and allergies as well as sensitization in children. In this follow-up report, sources of the PGEs in indoor air were investigated in 390 homes of pre-school age children in Sweden. Professional building inspectors examined each home for water damages, mold odour, building's structural characteristics, indoor temperature, absolute humidity and air exchange rate. They also collected air and dust samples. The samples were analyzed for four groups of volatile organic compounds (VOCs) and semi-VOCs (SVOCs), including summed concentrations of 16 PGEs, 8 terpene hydrocarbons, 2 Texanols, and the phthalates n-butyl benzyl phthalate (BBzP), and di(2-ethylhexyl)phthalate (DEHP). Home cleaning with water and mop ≥ once/month, repainting ≥ one room prior to or following the child's birth, and "newest" surface material in the child's bedroom explained largest portion of total variability in PGE concentrations. High excess indoor humidity (g/m³) additionally contributed to a sustained PGE levels in indoor air far beyond several months following the paint application. No behavioral or building structural factors, except for water-based cleaning, predicted an elevated terpene level in air. No significant predictor of Texanols emerged from our analysis. Overall disparate sources and low correlations among the PGEs, terpenes, Texanols, and the phthalates further confirm the lack of confounding in the analysis reporting the associations of the PGE and the diagnoses of asthma, rhinitis, and eczema, respectively.
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