[Deficiency of water channel AQP4 aggravates NMDA-induced brain injury in mice]

Related Experiment Video

Updated: Jun 2, 2026

Induction of Paralysis and Visual System Injury in Mice by T Cells Specific for Neuromyelitis Optica Autoantigen Aquaporin-4
09:29

Induction of Paralysis and Visual System Injury in Mice by T Cells Specific for Neuromyelitis Optica Autoantigen Aquaporin-4

Published on: August 21, 2017

Wen-Zhen Shi1, Chun-Zhen Zhao, Xue-Qin Huang

  • 1Department of Pharmacology, College of Medicine, Zhejiang University, Hangzhou 310058, China.

Zhejiang Da Xue Xue Bao. Yi Xue Ban = Journal of Zhejiang University. Medical Sciences
|April 14, 2011
PubMed
Abstract

More Related Videos

Assessing Spatial Memory Impairment in a Mouse Model of Traumatic Brain Injury Using a Radial Water Tread Maze06:09

Assessing Spatial Memory Impairment in a Mouse Model of Traumatic Brain Injury Using a Radial Water Tread Maze

Published on: July 17, 2017

Morris Water Maze Test for Learning and Memory Deficits in Alzheimer's Disease Model Mice07:02

Morris Water Maze Test for Learning and Memory Deficits in Alzheimer's Disease Model Mice

Published on: July 20, 2011

Related Concept Videos

Aquaporins01:25

Aquaporins

Aquaporins or AQPs are a family of integral membrane proteins whose primary function is to transport water, while some called aquaglyceroporins also transport glycerol. In addition, aquaporins have also been suspected to be involved in transporting volatile substances, such as carbon dioxide and ammonia, across membranes. Such AQPs that act as gas channels are often highly expressed in cells involved in the gaseous exchange, such as red blood cells, epithelial cells, and pulmonary capillaries.