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RNAscope for In situ Detection of Transcriptionally Active Human Papillomavirus in Head and Neck Squamous Cell Carcinoma
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Published on: March 11, 2014

HPV-associated oral warts.

L Feller1, R A G Khammissa, N H Wood

  • 1Department of Periodontology and Oral Medicine, School of Oral Health Sciences, Faculty of Health Sciences, University of Limpopo, Medunsa Campus, South Africa. Ifeller@ul.ac.za

SADJ : Journal of the South African Dental Association = Tydskrif Van Die Suid-Afrikaanse Tandheelkundige Vereniging
|May 26, 2011
PubMed
Summary
This summary is machine-generated.

Human papillomavirus (HPV) causes oral warts, which are benign epithelial lesions. This review examines the clinical and pathological features of these common oral lesions.

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Area of Science:

  • Oral pathology
  • Dermatology
  • Infectious diseases

Background:

  • Human papillomavirus (HPV) infects stratified squamous epithelial cells.
  • Oral HPV infections can be asymptomatic or linked to oral neoplasms.
  • Benign HPV-associated oral lesions include focal epithelial hyperplasia, papilloma, verruca vulgaris, and condyloma acuminatum, collectively termed oral warts.

Purpose of the Study:

  • To provide a clinico-pathological review of HPV-associated oral warts.
  • To summarize the clinical presentation and pathological findings of common oral warts.

Main Methods:

  • Literature review of clinico-pathological studies on HPV-associated oral warts.
  • Synthesis of existing data on prevalence, clinical features, and histopathology.

Main Results:

  • Oral warts are benign, usually asymptomatic lesions caused by HPV.
  • Prevalence is 0.5% in the general population, increasing in HIV-seropositive individuals (up to 5% and 23% on HAART).
  • Lesions can be persistent or regress spontaneously.

Conclusions:

  • HPV is a significant cause of benign oral lesions (oral warts).
  • Understanding the clinico-pathological features is crucial for diagnosis and management.
  • Higher prevalence in immunocompromised individuals highlights the role of immune status.