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Characterization of plasmids and plasmid-borne macrolide resistance from Lactobacillus sp. strain 100-33

L A Rinckel1, D C Savage

  • 1Department of Microbiology, University of Tennessee, Knoxville 37996.

Plasmid
|March 1, 1990
PubMed

Insights

Lactobacillus sp. strain 100-33

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Lactobacillus sp. strain 100-33 exhibits resistance to macrolides, lincosamides, and streptogramin B (MLSR).
  • This strain possesses major and minor plasmids, including the 18 kbp plasmid pLAR33.

Purpose of the Study:

  • To investigate the genetic basis of MLSR in Lactobacillus sp. strain 100-33.
  • To identify and characterize the specific plasmid(s) and genetic determinant(s) responsible for antibiotic resistance.

Main Methods:

  • Protoplast regeneration to derive antibiotic-sensitive strains.
  • Plasmid DNA isolation, cloning using EcoRV restriction enzyme into pBR322.
  • Transformation of Escherichia coli HB101 with cloned DNA.
  • Electroporation of resistant plasmid DNA into sensitive strains.
  • Endonuclease digestion and Southern blot analysis of plasmid DNA.

Main Results:

  • A sensitive derivative, strain ES1, was obtained by losing major plasmids including pLAR33.
  • The MLSR determinant was cloned from pLAR33 on a 3-kbp fragment and expressed resistance in E. coli.
  • The resistance determinant was localized to approximately 1 kbp and appears to have its own promoter.
  • Electroporation of strain ES1 with parental plasmid DNA restored MLSR phenotype and plasmid complement.

Conclusions:

  • The MLSR determinant is located on the plasmid pLAR33.
  • Major plasmids in strain 100-33 are multimeric, deleted forms of a single archetypal extrachromosomal element.
  • Understanding plasmid-mediated antibiotic resistance in Lactobacillus is crucial for food and probiotic applications.

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