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Nociceptive reflexes and the somatic dysfunction: a model.

R L Van Buskirk1

  • 1West Virginia School of Osteopathic Medicine, Lewisburg.

The Journal of the American Osteopathic Association
|September 1, 1990
PubMed
Summary
This summary is machine-generated.

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Pain-related sensory neurons and their reflexes cause somatic dysfunction, leading to mobility restrictions and changes in autonomic, visceral, and immunologic functions. This model highlights nociceptors as the source of these widespread effects.

Area of Science:

  • Neurology
  • Physiology
  • Immunology

Background:

  • Somatic dysfunction involves restricted mobility and associated autonomic, visceral, and immunologic changes.
  • Pain-related sensory neurons and their reflexes are implicated in these dysfunctions.
  • Musculoskeletal or visceral stress/damage activates nociceptors, leading to muscular guarding and autonomic responses.

Purpose of the Study:

  • To develop a model of somatic dysfunction centered on nociceptors and their reflexes.
  • To explain the origins of restricted mobility, autonomic, visceral, and immunologic alterations in somatic dysfunction.
  • To integrate Korr's neurologic model with an emphasis on nociceptive mechanisms.

Main Methods:

  • Development of a theoretical model based on known neurophysiologic principles.

Related Experiment Videos

  • Analysis of the role of nociceptors and their reflexes in musculoskeletal and visceral responses.
  • Examination of how sustained abnormal positioning affects connective tissues and reinforces dysfunction.

    • Main Results:

    • Pain-related sensory neurons and their reflexes are proposed as the primary drivers of somatic dysfunction.
    • Nociceptor activity, through muscular guarding and autonomic reflexes, causes restricted mobility.
    • Sustained abnormal positioning leads to connective tissue changes, solidifying the dysfunction and potentially causing visceral and immunologic alterations.

    Conclusions:

    • The proposed model emphasizes the nociceptor and its reflexes as the central mechanism in somatic dysfunction.
    • This model provides a unified explanation for the diverse changes (connective tissue, circulatory, visceral, immunologic) observed in somatic dysfunction.
    • Understanding this nociceptive model may inform new therapeutic strategies for somatic dysfunction.