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Biological activity of polyoma viral DNA in mice and hamsters

Journal of Virology
|March 1, 1979
PubMed

Insights

Polyoma viral DNA infectivity and tumor induction were studied in rodents. Supercoiled DNA caused infection in mice, but linearization reduced this. Early region cleavage significantly increased tumor development in hamsters.

Area of Science:

  • Virology
  • Molecular Biology
  • Oncology

Background:

  • Polyoma virus is a DNA tumor virus.
  • Understanding viral DNA's biological activity is crucial for virology and oncology research.

Purpose of the Study:

  • To evaluate the biological activity of polyoma viral DNA in mice and hamsters.
  • To compare the infectivity of supercoiled versus linearized viral DNA.
  • To assess the oncogenic potential of polyoma viral DNA following different enzymatic treatments.

Main Methods:

  • Parenteral administration of polyoma viral DNA (supercoiled and linearized) to mice.
  • Oral administration of polyoma viral DNA to mice.
  • Intraperitoneal inoculation of newborn hamsters with polyoma viral DNA.
  • Enzymatic cleavage of polyoma viral DNA using R.EcoRI and R.BamHI.

Main Results:

  • Parenteral administration of viral DNA was less efficient than virions for mouse infection.
  • Supercoiled viral DNA was infectious in mice, with mean infective doses of 10(-3) to 10(-4) microgram.
  • Linearization of viral DNA reduced infectivity in mice approximately fivefold.
  • Intraperitoneal inoculation of polyoma DNA I induced tumors in 10% of newborn hamsters.
  • Cleavage in the early region (R.EcoRI) increased tumor induction in hamsters to 50%.

Conclusions:

  • Supercoiled polyoma viral DNA exhibits infectivity in mice after parenteral administration.
  • Linearization of polyoma viral DNA significantly reduces its infectivity in mice.
  • Cleavage of polyoma viral DNA within the early region enhances its oncogenic potential in hamsters.

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