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Related Concept Videos

Equilibrium and Balance01:15

Equilibrium and Balance

The inner ear assumes dual functionalities of auditory perception and equilibrium maintenance. The vestibule is the organ responsible for balance. This organ contains mechanoreceptors, specifically hair cells, endowed with stereocilia, which aid in deciphering information regarding the position and motion of our heads. Two intrinsic components, the utricle and saccule, help perceive head position, while the semicircular canals track head movement. Neurological messages initiated in the...
Transient Ischemic Attack l: Introduction01:26

Transient Ischemic Attack l: Introduction

A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...
Major Somatic Sensory Pathways01:28

Major Somatic Sensory Pathways

Sensory impulses related to touch, pressure, vibration, and proprioception from various body parts, such as the limbs, trunk, neck, and posterior head, travel to the cerebral cortex through the posterior column-medial lemniscus pathway. The pathway’s name derives from the two white-matter tracts that convey the impulses: the spinal cord's posterior column and the brainstem's medial lemniscus. First-order sensory neurons extend their axons into the spinal cord, forming the posterior columns...
Hemorrhagic Stroke l: Introduction01:17

Hemorrhagic Stroke l: Introduction

A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
Stroke: Introduction and Types01:29

Stroke: Introduction and Types

A stroke is an acute neurological event caused by the sudden disruption of cerebral blood flow, leading to rapid loss of neuronal function. Neurons depend on continuous oxygen and glucose supply, so even brief interruptions can cause irreversible injury within minutes. Strokes are classified into ischemic and hemorrhagic types.Ischemic StrokeIschemic strokes are most common and occur due to arterial occlusion, depriving brain tissue of oxygen and nutrients. This leads to energy failure, ionic...
Cerebral Edema ll: Pathophysiology01:22

Cerebral Edema ll: Pathophysiology

Vasogenic edema is a major form of cerebral edema characterized by abnormal accumulation of fluid in the brain’s extracellular space due to disruption of the blood–brain barrier (BBB). The BBB is a specialized structure composed of endothelial cells connected by tight junctions, supported by astrocytic endfeet and a basement membrane. Under normal conditions, it tightly regulates the movement of ions, proteins, and solutes between the bloodstream and brain parenchyma. When this barrier loses...

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Related Experiment Video

Updated: May 25, 2026

Using Eye-tracking to Assess the Relative Importance of Visual and Vestibular Input to Subcortical Motion Processing in the Roll Plane
07:24

Using Eye-tracking to Assess the Relative Importance of Visual and Vestibular Input to Subcortical Motion Processing in the Roll Plane

Published on: August 22, 2025

[Dizziness/vertigo caused by small cerebellar/brainstem strokes].

Ken Johkura1

  • 1Department of Neurology, Hiratsuka Kyosai Hospital.

Rinsho Shinkeigaku = Clinical Neurology
|January 27, 2012
PubMed
Summary

Small cerebellar strokes can mimic vestibular disorders, causing isolated dizziness and specific nystagmus patterns. These eye movement abnormalities, particularly horizontal nystagmus and direction-changing apogeotropic positional nystagmus, are enhanced when patients lie on their unaffected side.

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Published on: March 3, 2023

Related Experiment Videos

Last Updated: May 25, 2026

Using Eye-tracking to Assess the Relative Importance of Visual and Vestibular Input to Subcortical Motion Processing in the Roll Plane
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Using Eye-tracking to Assess the Relative Importance of Visual and Vestibular Input to Subcortical Motion Processing in the Roll Plane

Published on: August 22, 2025

Measuring the Influence of Magnetic Vestibular Stimulation on Nystagmus, Self-Motion Perception, and Cognitive Performance in a 7T MRT
08:57

Measuring the Influence of Magnetic Vestibular Stimulation on Nystagmus, Self-Motion Perception, and Cognitive Performance in a 7T MRT

Published on: March 3, 2023

Area of Science:

  • Neurology
  • Ophthalmology
  • Neuroscience

Context:

  • Cerebellar strokes can present with symptoms mimicking peripheral vestibular disorders.
  • Distinguishing cerebellar strokes from vestibular neuritis and benign paroxysmal positional vertigo is clinically important.

Purpose:

  • To describe the characteristics of horizontal nystagmus and direction-changing apogeotropic positional nystagmus in small cerebellar strokes.
  • To explore the potential mechanism behind the enhancement of these nystagmus patterns.

Summary:

  • Small cerebellar strokes can cause isolated dizziness and horizontal nystagmus directed toward the lesion side.
  • This horizontal nystagmus, along with direction-changing apogeotropic positional nystagmus, is enhanced when the patient lies on the non-affected side.
  • A proposed mechanism involves the disinhibition of otolith-ocular responses.

Impact:

  • Improves diagnostic accuracy for cerebellar strokes presenting with vestibular symptoms.
  • Highlights the significance of positional nystagmus testing in the neurological examination.
  • Contributes to understanding the pathophysiology of vestibular eye movement disorders.