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Delayed-onset bilateral abducens paresis after head trauma
Pravin Salunke1, Amey Savardekar, Sukumar Sura
1Department of Neurosurgery, PGIMER, Chandigarh, India. salunke@yahoo.co.uk
Indian Journal of Ophthalmology
|March 27, 2012
Summary
Delayed abducens nerve palsy after head injury is rare. This study presents two cases, highlighting varied causes like subdural hematoma and potential edema, emphasizing the need for evaluation of elevated intracranial pressure.
Area of Science:
- Neuroscience
- Ophthalmology
- Traumatology
Background:
- Bilateral sixth nerve (abducens nerve) paresis is a recognized, albeit uncommon, consequence of closed head injury.
- Delayed-onset presentation of this condition following traumatic brain injury is exceptionally rare.
Observation:
- Two cases of delayed-onset bilateral abducens nerve paresis following closed head injury are presented.
- The first case manifested 2 weeks post-injury, attributed to a chronic subdural hematoma.
- The second case appeared 3 days post-injury, with edema or ischemia of nerve-housing soft tissues speculated as the cause.
Findings:
- The temporal delay in the onset of bilateral abducens paresis appears to correlate with the underlying etiology.
- Mechanisms beyond direct trauma, such as increased intracranial pressure, may contribute to this delayed paresis.
- Chronic subdural hematoma was identified as a cause in one patient.
Implications:
- Delayed-onset bilateral abducens paresis post-head injury warrants thorough investigation due to potentially treatable causes.
- Evaluation for elevated intracranial pressure should be considered in such cases.
- Understanding varied injury mechanisms is crucial for managing post-traumatic cranial nerve palsies.
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