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Related Experiment Video

Updated: May 22, 2026

A Murine Model of Ischemic Retinal Injury Induced by Transient Bilateral Common Carotid Artery Occlusion

A Murine Model of Ischemic Retinal Injury Induced by Transient Bilateral Common Carotid Artery Occlusion

Published on: November 12, 2020

Branch retinal arterial occlusion.

S Subedi¹, C Shrestha

¹Department of Ophthalmology, NAMS, Nepal Eye Hospital, Nepal. ssubedi63@rediffmail.com

Kathmandu University Medical Journal (KUMJ)

|May 22, 2012

Summary

This summary is machine-generated.

A young woman experienced sudden vision loss due to Branch Retinal Arterial Occlusion, a rare event in individuals without typical risk factors. Prompt observation led to vision recovery, though visual field defects persisted.

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In Vivo Vascular Injury Readouts in Mouse Retina to Promote Reproducibility

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Last Updated: May 22, 2026

A Murine Model of Ischemic Retinal Injury Induced by Transient Bilateral Common Carotid Artery Occlusion

A Murine Model of Ischemic Retinal Injury Induced by Transient Bilateral Common Carotid Artery Occlusion

Published on: November 12, 2020

In Vivo Vascular Injury Readouts in Mouse Retina to Promote Reproducibility

In Vivo Vascular Injury Readouts in Mouse Retina to Promote Reproducibility

Published on: April 21, 2022

Area of Science:

Ophthalmology
Cardiology
Vascular Medicine

Background:

Retinal arterial occlusion is a critical condition impacting vision, often due to delayed diagnosis and macular damage.
Branch Retinal Arterial Occlusion (BRAO) is less common, particularly in young adults without systemic risk factors.

Observation:

A 22-year-old female presented with painless vision loss and a visual field defect in her right eye.
Fundus examination revealed retinal whitening in the inferotemporal arcade, encroaching on the foveolar avascular zone.
Echocardiography identified Grade II Mitral Regurgitation as a potential underlying factor.

Findings:

Despite normal initial fundus fluorescence angiography, the patient had a visual field defect.
Initial visual acuity of 6/60 in the affected eye improved to 6/6 within two days.
The visual field defect remained unchanged post-treatment.

Implications:

This case highlights the importance of considering BRAO in young individuals presenting with visual disturbances, even without typical risk factors.
Mitral regurgitation may be a contributing factor in some cases of BRAO.
While visual acuity can recover, persistent visual field defects underscore the potential long-term impact of BRAO.