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Modulation of oestrogen receptor activity by oestrogens and anti-oestrogens

S Green1

  • 1Imperial Chemical Industries, Central Toxicology Laboratory, Macclesfield, Cheshire, England.

Insights

The oestrogen receptor (ER) is a transcription factor regulating gene expression. Tamoxifen, an anti-oestrogen, acts as a weak ER agonist, with its effects explained by a molecular model.

Area of Science:

  • Molecular Endocrinology
  • Genetics
  • Pharmacology

Background:

  • The oestrogen receptor (ER) belongs to a supergene family, acting as a ligand-activated transcription factor.
  • ER modulates gene expression by binding to oestrogen response elements in DNA.
  • Other family members exist with unidentified ligands.

Purpose of the Study:

  • To identify regions of the ER responsible for hormone-binding, DNA-binding, and transcriptional activation.
  • To present a model explaining the cell and gene-specific effects of anti-oestrogens like tamoxifen.

Main Methods:

  • Identification of functional regions within the oestrogen receptor.
  • Development of a molecular model for oestrogen action and anti-oestrogen effects.

Main Results:

  • Specific regions of the ER mediating hormone-binding, DNA-binding, and transcriptional activation have been identified.
  • Tamoxifen (Nolvadex) exhibits weak oestrogen agonist activity.

Conclusions:

  • A molecular model elucidates the mechanism of oestrogen action.
  • This model explains the complex, cell-specific effects of anti-oestrogens on gene expression.

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