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[Rapidly progressing quadriparesis secondary to licorice (souss) intoxication]
Hatem Korri1, Adnan Awada, Wadih Baajour
1Services de Neurologie, Hôtel-Dieu de France. hatem_korri2@hotmail.com
Le Journal Medical Libanais. the Lebanese Medical Journal
|August 28, 2012
Summary
Excessive licorice consumption can cause severe muscle weakness due to hypokalemia from pseudohyperaldosteronism. Prompt electrolyte correction rapidly resolves symptoms, highlighting the importance of recognizing licorice toxicity.
Area of Science:
- Endocrinology
- Toxicology
- Neurology
Background:
- Licorice consumption is linked to pseudohyperaldosteronism and hypokalemia.
- Severe muscle weakness is a rare but serious complication of licorice toxicity.
Observation:
- A 62-year-old man presented with progressive, four-limb muscle weakness.
- Laboratory results indicated severe hypokalemia and primary pseudohyperaldosteronism.
Findings:
- The patient's condition was diagnosed as licorice intoxication causing secondary pseudohyperaldosteronism.
- Electrolyte disturbances were identified as the direct cause of his muscle weakness.
Implications:
- In regions with high licorice beverage consumption, clinicians should consider licorice toxicity in cases of muscle weakness with hypokalemia.
- Rapid correction of electrolyte imbalances is crucial for patient recovery.
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