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Updated: May 17, 2026

An Effective Inoculation Method for Phytophthora capsici on Black Pepper Plants

An Effective Inoculation Method for Phytophthora capsici on Black Pepper Plants

Published on: September 16, 2022

A pox upon your house.

Nafiseh Hashemi¹, Jason Zhang, John Volpi

¹Department of Ophthalmology, Houston Methodist Hospital, Houston, Texas, USA.

Survey of Ophthalmology

|November 13, 2012

Summary

This summary is machine-generated.

Herpes zoster ophthalmicus sine herpete, a rare condition without a rash, can lead to severe neurological complications like encephalitis and cerebral infarction, even with treatment. This case highlights the importance of considering HZV in unexplained neurological symptoms.

Keywords:

cerebral infarction encephalitis herpes zoster ophthalmicus herpes zoster sine herpete uveomeningeal syndrome

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Area of Science:

Neurology
Infectious Diseases
Ophthalmology

Background:

Herpes zoster ophthalmicus (HZO) typically involves a rash along the ophthalmic nerve.
Herpes zoster sine herpete (HZSH) presents with ophthalmic symptoms but lacks a rash.
Central nervous system (CNS) involvement in HZO is known, but encephalitis and cerebral infarction are rare.

Observation:

A case of presumed unilateral HZSH presented with trigeminal pain and uveitis.
The patient developed encephalitis and bilateral cerebral infarctions.
These severe complications occurred despite treatment with acyclovir and corticosteroids.

Findings:

Polymerase chain reaction (PCR) testing on cerebrospinal fluid confirmed Herpes zoster virus (HZV) diagnosis.
HZV infection can manifest as HZSH with severe, atypical CNS complications.
This case underscores the potential for HZV to cause significant neurological damage without a cutaneous rash.

Implications:

Early recognition and aggressive management of HZSH are crucial to prevent devastating neurological sequels.
Neurologists and ophthalmologists should consider HZV in the differential diagnosis of unexplained trigeminal pain, uveitis, encephalitis, and cerebral infarction.
Further research is needed to understand the pathogenesis and optimize treatment strategies for HZSH with CNS involvement.