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Endovascular management of may-thurner syndrome
Wael Ibrahim1, Zakareya Al Safran, Hosam Hasan
1Medical Imaging Department, King Fahad Specialist Hospital, Dammam, Saudi Arabia.
Insights
May-Thurner syndrome, a cause of deep vein thrombosis (DVT), involves left iliac vein compression. Endovascular treatment is increasingly recommended over anticoagulation alone for better outcomes.
Area of Science:
- Vascular Surgery
- Interventional Radiology
- Cardiovascular Medicine
Background:
- May-Thurner syndrome (MTS), or iliac vein compression syndrome, links deep vein thrombosis (DVT) to left iliac vein compression by the right common iliac artery.
- Traditional treatment with anticoagulation alone is insufficient, as it doesn't resolve the clot or the underlying compression, leading to recurrent DVT or post-thrombotic syndrome.
Observation:
- A 53-year-old male patient presented with May-Thurner syndrome.
- The patient's condition was managed using endovascular treatment, which addresses both the mechanical compression and the thrombus burden.
Findings:
- Endovascular management, including stent placement and chemical thrombolysis, actively treats the mechanical compression and thrombus in May-Thurner syndrome.
- This approach offers a more comprehensive solution compared to anticoagulation therapy alone.
Implications:
- Endovascular treatment is emerging as a preferred front-line therapy for May-Thurner syndrome, potentially reducing recurrence rates of DVT and post-thrombotic syndrome.
- This case highlights the successful application of endovascular techniques in managing MTS, supporting its role in clinical practice.
Abstract:
May-Thurner syndrome or iliac vein compression syndrome is associated with deep vein thrombosis (DVT) resulting from chronic compression of the left iliac vein against lumbar vertebrae by the overlying right common iliac artery. Historically, May-Thurner syndrome has been treated with anticoagulation therapy. However, this therapy can be problematic when given alone, because it prevents the propagation of the thrombus without eliminating the existing clot. Furthermore, it does not treat the underlying mechanical compression. Consequently, syndrome who was managed by anticoagulation therapy alone, there is a significant chance that the patient will develop recurrent deep vein thrombosis or post thrombotic syndrome or both. Recently, both retrospective and prospective studies have suggested that endovascular management should be front-line treatment; endovascular management actively treats both the mechanical compression with stent placement and the thrombus burden with chemical dissolution. We report our case of 53 years old male patient with May Thurner syndrome who managed by endovascular treatment.
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