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Related Concept Videos

Cellular Injury IV: Necrosis01:16

Cellular Injury IV: Necrosis

Necrosis is a form of irreversible cell death caused by severe injury such as ischemia, toxins, or trauma. Unlike programmed cell death, it is an uncontrolled, pathological process that typically provokes inflammation in surrounding tissues.Pathophysiologic ChangesNecrosis begins when cells sustain critical damage, leading to swelling of organelles, particularly mitochondria, and rapid ATP depletion. As energy levels decline, membrane ion pumps fail, leading to calcium influx and eventually,...
Necrosis01:16

Necrosis

Necrosis is considered as an “accidental” or unexpected form of cell death that ends in cell lysis. The first noticeable mention of “necrosis” was in 1859 when Rudolf Virchow used this term to describe advanced tissue breakdown in his compilation titled “Cell Pathology”.
Morphological Manifestations of Necrosis
Necrotic cells show different types of morphological appearance depending on the type of tissue and infection. In coagulative necrosis, cells become anucleated and die, but their...
Endocarditis II: Clinical Features of Infective Endocarditis01:25

Endocarditis II: Clinical Features of Infective Endocarditis

Endocarditis can present various clinical features depending on the causative organism and the patient's underlying health conditions. Initially, the clinical features of infective endocarditis develop gradually, presenting with nonspecific symptoms that can be easily mistaken for other illnesses.General SymptomsEarly symptoms of infective endocarditis are fever, chills, weakness, malaise, fatigue, and weight loss. These symptoms reflect the systemic nature of the infection and the body's...
Bacterial Meningitis I: Introduction01:22

Bacterial Meningitis I: Introduction

Bacterial meningitis is a severe, life-threatening inflammation of the meninges, particularly the pia mater and arachnoid mater, affecting the subarachnoid space, ventricles, and cerebrospinal fluid (CSF). If untreated, it can lead to significant neurological complications or death.Causative AgentsCommon pathogens vary with age and immune status. In adults, major organisms include Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. Streptococcus agalactiae (group B...
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Cytotoxic Edema: Pathophysiology

Cytotoxic edema is a form of cerebral edema characterized by intracellular swelling of neurons, astrocytes, and other glial cells. It develops when the mechanisms responsible for maintaining ionic gradients across the cell membrane become impaired. Under normal physiological conditions, the sodium–potassium ATPase actively transports sodium ions out of the cell and potassium ions into the cell, preserving osmotic balance and enabling electrical signaling. This pump requires a continuous supply...
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Related Experiment Videos

[Cervico-facial necrotizing fasciitis].

Patrice Tran Ba Huy1

  • 1Service d'oto-rhino-laryngologie, hôpital Lariboisière, AP-HP, 75009 Paris, France. patrice.tran-ba-huy@lrb.aphp.fr

La Revue Du Praticien
|August 9, 2013
PubMed
Summary

Cervical fasciitis, a severe neck infection, demands urgent multidisciplinary care. Prompt CT scans, surgical drainage, and intensive medical support are crucial for survival and minimizing complications.

Area of Science:

  • Otolaryngology
  • Infectious Diseases
  • Surgical Critical Care

Background:

  • Cervical fasciitis is a severe, life-threatening infection of the neck's fascial planes.
  • Often originating from benign pharyngeal or dental infections, it can rapidly progress to extensive necrosis and mediastinal involvement.
  • It constitutes a critical Ear, Nose, and Throat (ENT) emergency requiring immediate, expert intervention.

Purpose of the Study:

  • To highlight the critical nature of cervical fasciitis.
  • To emphasize the necessity of prompt diagnosis and multidisciplinary management.
  • To outline essential treatment protocols and expected patient outcomes.

Main Methods:

  • Review of clinical presentation and management of cervical fasciitis.

Related Experiment Videos

  • Emphasis on urgent referral to tertiary centers for advanced imaging and surgical intervention.
  • Discussion of intensive medical care requirements for managing complications.

    • Main Results:

    • High mortality rate (5-10%) and significant morbidity, including pneumopathy (1/3), hemodynamic failure, and mediastinitis (1/2).
    • Prolonged intensive care unit stays with extended mechanical ventilation, intubation, and tracheostomy durations.
    • Functional and esthetic sequelae observed in approximately 50% of patients.

    Conclusions:

    • Cervical fasciitis necessitates vigilant recognition of early signs of severe sepsis from head and neck infections.
    • Immediate CT imaging, surgical drainage, and comprehensive intensive care are vital for patient survival.
    • Inadequate initial treatment and self-medication may contribute to disease progression.