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Related Concept Videos

Barrett Esophagus-I: Introduction01:21

Barrett Esophagus-I: Introduction

Barrett's esophagus is a medical condition where the esophageal mucosa is significantly damaged by stomach acid or other digestive fluids, often due to long-term exposure associated with gastroesophageal reflux disease (GERD). In GERD, a weakened or abnormally relaxed lower esophageal sphincter allows stomach acid to flow persistently into the esophagus.
This constant acid exposure transforms the esophagus's pink mucosal lining (stratified squamous epithelium) into a type of lining more similar...
Barrett Esophagus-II: Clinical Manifestations and Management01:21

Barrett Esophagus-II: Clinical Manifestations and Management

Individuals with Barrett's esophagus are often asymptomatic, but they may experience symptoms commonly associated with GERD, such as heartburn and acid regurgitation. Additional symptoms can include difficulty swallowing, chest pain, unintentional weight loss, blood in the stool (which may appear black, tarry, or bloody), and episodes of vomiting.
To diagnose Barrett's esophagus, healthcare providers often recommend an endoscopy for those showing symptoms of acid reflux. The procedure entails...
Esophageal Achalasia01:27

Esophageal Achalasia

Esophageal achalasia is a chronic neurogenic disorder characterized by impaired relaxation of the lower esophageal sphincter (LES) and absent or ineffective peristalsis in the distal esophagus. This leads to a functional obstruction without a physical blockage, despite significant disruption of esophageal motility.EtiologyAchalasia is caused by degeneration of the myenteric (Auerbach's) plexus, specifically the loss of inhibitory ganglion cells that produce vasoactive intestinal peptide (VIP)...
Esophageal Strictures-I: Introduction01:30

Esophageal Strictures-I: Introduction

Esophageal strictures involve abnormal narrowing or tightening of the esophagus. They vary in length and severity, ranging from mild constriction to complete obstruction, and are classified as benign (noncancerous) or malignant (cancerous).
Etiology
The primary cause of esophageal strictures is long-standing gastroesophageal reflux disease (GERD), accounting for about 70 to 80% of adult cases. Chronic acid reflux can lead to injury and scarring of the esophageal lining, culminating in...
Esophagus01:24

Esophagus

The esophagus, a muscular conduit linking the pharynx and stomach, measures roughly 10 inches (25.4 cm) and sits behind the trachea. It remains collapsed when not swallowing. The esophagus follows a predominantly straight path through the thoracic mediastinum and enters the abdominal cavity through a diaphragmatic opening known as the esophageal hiatus.
The movement of edibles from the pharynx into the esophagus is facilitated by the upper esophageal sphincter, which is formed primarily by the...
Esophageal Varices-I: Introduction01:24

Esophageal Varices-I: Introduction

Esophageal varices are dilated, tortuous veins which are found mainly in the submucosa of the lower esophagus but which may also appear higher up or extend into the stomach. They develop due to increased pressure in the portal venous system, often as a result of liver cirrhosis. This condition scars and damages the liver, impeding normal blood flow through the portal vein. To compensate, blood seeks alternative pathways, forming fragile new vessels (varices) in the esophagus and stomach. These...

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Related Experiment Video

Updated: May 8, 2026

Tissue-Engineered Graft for Circumferential Esophageal Reconstruction in Rats
08:56

Tissue-Engineered Graft for Circumferential Esophageal Reconstruction in Rats

Published on: February 10, 2020

[Black esophagus].

A Hermet1, O Lidove1, S Grimbert2

  • 1Service de médecine interne et rhumatologie, hôpital de la Croix Saint-Simon, 125, rue d'Avron, 75020 Paris, France.

La Revue De Medecine Interne
|August 17, 2013
PubMed
Summary
This summary is machine-generated.

Acute esophageal necrosis, or black esophagus, is a rare condition. This case highlights its occurrence following hip replacement due to hemodynamic compromise, with favorable outcomes after treatment.

Keywords:
Acute esophageal necrosisBlack esophagusHematemesisHématémèseNécrose aiguë de l’œsophageŒsophage noir

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Using the Endoscope for Endobronchial Ultrasound in the Esophagus
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Using the Endoscope for Endobronchial Ultrasound in the Esophagus

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Related Experiment Videos

Last Updated: May 8, 2026

Tissue-Engineered Graft for Circumferential Esophageal Reconstruction in Rats
08:56

Tissue-Engineered Graft for Circumferential Esophageal Reconstruction in Rats

Published on: February 10, 2020

ADSC-sheet Transplantation to Prevent Stricture after Extended Esophageal Endoscopic Submucosal Dissection
05:57

ADSC-sheet Transplantation to Prevent Stricture after Extended Esophageal Endoscopic Submucosal Dissection

Published on: February 10, 2017

Using the Endoscope for Endobronchial Ultrasound in the Esophagus
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Using the Endoscope for Endobronchial Ultrasound in the Esophagus

Published on: November 21, 2023

Area of Science:

  • Gastroenterology
  • Cardiovascular Medicine
  • Surgical Complications

Background:

  • Acute esophageal necrosis, termed black esophagus, is an uncommon clinical diagnosis.
  • It is often associated with critical illness, hypoperfusion, and significant physiological stress.

Observation:

  • A 72-year-old male with multiple comorbidities experienced hematemesis and epigastric pain post-total hip replacement.
  • Gastric endoscopy revealed acute esophageal necrosis, indicative of severe hypoperfusion.
  • The patient had a history of coronary heart disease, peripheral artery disease, diabetes, and hypertension.

Findings:

  • Esophageal necrosis developed secondary to hemodynamic compromise after major orthopedic surgery.
  • Treatment involved intravenous proton pump inhibitors, parenteral nutrition, and blood transfusion.
  • Endoscopic follow-up demonstrated complete resolution of esophageal necrosis within four days.

Implications:

  • This case underscores the potential for esophageal necrosis in debilitated patients experiencing hypoperfusion and stress, even after elective surgery.
  • Prompt medical management can lead to favorable outcomes, even in patients with significant comorbidities.
  • Awareness of this rare complication is crucial for timely diagnosis and intervention in post-operative patients presenting with gastrointestinal symptoms.