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Philodendron--an infant death.

M S McIntire1, J R Guest, J F Porterfield

  • 1Creighton University School of Medicine, Omaha, Nebraska.

Journal of Toxicology. Clinical Toxicology
|January 1, 1990
PubMed
Summary
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Philodendron plant ingestion caused severe esophageal lesions and dysphagia in an infant. The esophageal damage led to vagotonia and unexpected sudden death.

Area of Science:

  • Toxicology
  • Pediatric Gastroenterology
  • Plant Toxicology

Background:

  • Philodendron species (Araceae family) contain insoluble calcium oxalate crystals.
  • Ingestion of toxic plants can lead to severe gastrointestinal and systemic complications in children.
  • Oropharyngeal and esophageal injuries are known complications of certain plant ingestions.

Observation:

  • An 11-month-old infant experienced oropharyngeal erosions and dysphagia after chewing Philodendron leaves.
  • Esophageal erosions and a cricoid-level esophageal stricture were diagnosed 16 days post-ingestion.
  • The infant developed unexpected sudden death on day 17.

Findings:

  • The clinical presentation suggests a severe inflammatory response and tissue damage from Philodendron ingestion.

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  • Esophageal lesions, including stricture formation, were confirmed.
  • The cause of death was attributed to vagotonia secondary to the esophageal lesions.

    • Implications:

    • This case highlights the potential for severe toxicity from Philodendron plant ingestion in children.
    • Prompt medical evaluation and management are crucial for pediatric plant ingestions.
    • Vagotonia secondary to esophageal lesions is a potential, life-threatening complication.